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PEAK 485
Exercise Test and Prescription
The Heart of the Matter
Cardiovascular health is key
Poor blood supply = damage / death
Cardiac muscle is the ultimate slow twitch fiber
Cardiac function predicts health and performance
Cardiology
Cardiac Function
Two cycle pump
Receives blood from lungs and body
Sends blood to lungs and body
Closed circuit (“leaky” / volume changes)
Arterial (systemic) vs. venous vascular
systems
Cardiac Anatomy
Four Chambers
2 Atria and 2 Ventricles
Left side vs. right side
Key Features
Left Ventricle is primary pump (systemic circulation)
Right Atrium
Sinoatrial node (SA node)
Cardiac Anatomy
Great Vessels
Sup. & Inf. Vena Cava
Pulmonary Arteries
Pulmonary Veins
Aorta
Blood source for coronary arteries
Cardiac Diagram
Cardiac Anatomy
Coronary Arteries (lesser vessels)
Left Coronary Artery
Anterior Descending Art. (ant. supply) (LAD)
Left Circumflex Art. (lat. left V. supply)
Right Coronary Art. (right A. & V.)
Runs infer. to post.
AV node supply
Each supplies given part of myocardium with
blood flow (nutrients and oxygen).
Left
Circumflex
Cardiac Anatomy
Coronary Arteries (lesser vessels)
Capillary Beds
Site of oxygen diffusion
Capillary Beds
Cardiac Anatomy
Heart Layers
Endocardium
Myocardium*
Epicardium
Pericardium
Cardiac Anatomy
Myocardium – Heart Muscle
Myocytes
Intercalated discs
Cell orientation
Nervous stimulation
Sympathetic system
Parasympathetic system
Cell communication
Cardiac Anatomy
Conduction System
SA node
AV node
Bundle of His
Bundle Branches (septal to apex to walls)
Left Bundle Branch (LBB)
o Right Bundle Branch (RBB)
Purkinje fibers
Cardiac Physiology
Cardiac Physiology
Properties of myocardial cells:
Contractility
Strength of contraction (tension) can vary
Extensibility / Elasticity
Cardiac Physiology
Properties of myocardial cells cont’d
Excitability / Irritability
Can be electrically or chemically stimulated
Balance of chemicals creates membrane potential
Disruption of chemical balance leads to stimulation
Ionotropic effect – speed up
Ca
Cardiac Physiology
Automaticity
Leaky / channels
Spontaneous depolarization.
Pacemaker Cells (SA Node)
This tissue normally initiates the cardiac cycle.
Cardiac Physiology
Conductivity
Action potential travels to other cells within the
atria (or other cells) causing them to contract.
Cardiac Physiology
Properties of myocardial cells cont’d
Extensibility / Elasticity
Electrical Conduction
AV Node – Delay station for electrical impulse. Delay
allows atrial contraction to complete before ventricles
begin.
Bundle of His – Transmits electrical impulse across the
annulus fibrosis (cartilage framework of atrioventricular
valves).
Bundle Branches (and fascicles) – conduct impulse
rapidly throughout ventricles.
Note: Process allows for sequential contraction of the
myocardium to pump blood effectively in the proper
direction.
Questions So Far?
ECG Principles
ECG Principles
ECG Principles
ECG Paper
Standard paper speed = 25 mm/s
Small and large boxes
Horizontally - time
Small boxes represent .04 sec (40 msec)
Large boxes represent .20 sec (200 msec)
Vertically – strength of electrical activity (voltage)
Small boxes represent .1 mV
Large boxes represent .5 mV
ECG Principles
Electrical & Mechanical Events
P wave
Atrial depolarization
Atrial contraction begins midway
QRS wave
Ventricular depolarization
Ventricular contraction begins midway/late
T wave
Ventricular repolarization
Ventricular relaxation
ECG Tracing Diag.
ECG Principles
Timing of Electrical Events
PR interval – atrial depol. .12 - .20 sec.
PR segment – AV/His depol. – isoelectric
QRS complex - < .12 sec
ST segment –vent. cells depolarized - isoelectric
QT interval - < .36 sec
P-P / R-R - Atr. & Vent. rate dependent cycles
ECG Tracing Diag.
Timing changes (fast or slow) =
Normal response
Abnormal response
ECG Waves and Electrodes
Reference point is the positive electrode.
Depolarization wave moves toward the positive
electrode, recorded depolarization wave is
positive.
Depolarization wave moves away from the
positive electrode, recorded depolarization wave is
negative.
ECG Waves and Electrodes
If the positive electrode is perpendicular (sits
across the axis) to the depolarization wave, the
recording is biphasic (both positive and negative).
ECG Waves and Electrodes
Limb Leads:
I, II, III: each has two electrodes (bipolar
leads – one positive; one negative)
Limb Leads Diag.
ECG Waves and Electrodes
Limb Leads:
Augmented Voltage Leads (AVR, AVL, AVF)
- each has a positive electrode but shares an
averaged (common) negative pole made up of
the other electrodes.
Augmented Leads Diag.
ECG Waves and Electrodes
Limb Leads:
These six leads view the electrical activity in
the vertical plane.
ECG Waves and Electrodes
Hexaxial Reference System
Measured in degrees starting with positive
side of Lead I being 0°.
Rotation clockwise from 0° is positive;
counterclockwise is negative.
Hexaxial Diag.
AVR
AVL
AVF
ECG Waves and Electrodes
Precordial Leads
V1, V2, V3, V4, V5, V6 – each is a positive
electrode with the entire body serving as the
negative pole.
These six leads view the electrical activity in
the horizontal plane.
Precordial Leads Diag.
Heart level Xsect. Diag.
Cross Section of the Heart at Level of T4 - T5 (Image from the Visible
Human Project-NLM)
V1
Horiz. Hexaxial Diag.
V6
Cross Section of the Heart at Level of T4 - T5 (Image from the Visible
Human Project-NLM)
Vectors and Waveforms
Vector = average electrical activity – direction
Atria – downward & leftward
AV Node & BB’s – not strong enough to detect
Septum – leftward
Ventricles – downward & leftward
V. Repolarization – same direction
Vector Diag.
P
Vectors and Waveforms
Remember
– Waveform changes for each lead
– Tallest toward + electrode
– Inverted away from + electrode
– Biphasic perpendicular to line of + to –
– View of multiple leads tells direction of
depolarization
Questions So Far?
ECG Interpretation
Basic Questions?
ECG Interpretation
What do you assess?
What order do you follow?
What meaning does a deviation have?
What should be done?
ECG Interpretation
Rate
Rhythm
Axis
Hypertrophy
Infarction
ECG Interpretation
Rate
Methods to determine heart rate:
1500/# small boxes between R waves.
o
o
Most accurate; best for fast rhythms.
Question: How much time is one small box?
300/# large boxes between R waves.
o
o
o
Good for slow rhythms.
300, 150, 100, 75, 60, 50 rule
Question: How much time is one large box?
# R waves in 6.0 s (30 large boxes) x 10 (3 sec. marks)
o
Good for irregular rhythms.
ECG Interpretation
Quick Visual: Rate at each heavy line (large box)
0 300 150 100 75 60 50 43 38 33 30
350
250
(4th)
(6th)
Heart rate determination
ECG Interpretation
Ventricular Rate
Atrial Rate
Nodal Rate
What’s The Rate?
What’s The Rate?
What’s The Rate?
ECG Interpretation
Source? – atria, junction, ventricles?
Normal rate = 60-80 bpm
Tachycardia = > 100 bpm
Bradycardia = < 60 bpm
Flutter = > 250 – 350 “bpm”
– Atrial source – “sawtooth” appearance
– Ventricular pacing is not connected
Fibrillation = > 350 “bpm”
– Either atrial or ventricular source – “bag of worms” – no cardiac output
– V. Fib. = immediate syncope, asystole is soon!!!!
What’s the rate? – What’s it called?
Sinus tachycardia
What’s the rate? – What’s it called?
Anything else you can see?
Sinus bradycardia
ECG Interpretation
Rhythm
Lead II is often best to observe rhythm.
Analysis:
1. Determine regularity of rhythm.
Index card method
Calipers
2. Determine rate.
3. Examine P waves:
Appearance is normal and consistent
P before each QRS; one P wave for each QRS
ECG Interpretation
Rhythm
4. Measure PR interval
Normally < .20 s
5. QRS complex width:
normally narrow (< .12 s)
ECG Interpretation
Arrhythmias
Four basic types of arrhythmias:
1. Sinus origin
2. Ectopic rhythms
3. Conduction blocks
4. Preexcitation syndromes
ECG Interpretation
Arrhythmias
1.
Sinus Origin
Sinus bradycardia (< 60 bts/min)
Sinus tachycardia (>100 bts/min at rest)
Sinus Arrhythmia:
Could be normal rate but rate changes (does not remain
regular)
o
Sinus Arrest
o
Escape beats
ECG Interpretation
Arrhythmias
2.
Ectopic Rhythms – originate outside of the SA node
•
Supraventricular - Atrial focus = 60-80
•
•
When SA node does not fire
Increased irritability of atrial tissue
AV node – does no pacemaking – holds signal
Junctional focus = 40-60
•
•
•
•
•
Idiojunctinal – Bundle of His
No signal from SA or atria
Retrograde depolarization (P wave?)
ECG Interpretation
Arrhythmias
2.
Ectopic Rhythms – originate outside of the SA node
•
Ventricular Focus = 20-40
•
•
•
•
Idioventricular
No signal from SA, atria, or junction
Larg, wide QRS wave
P wave? - none
ECG Interpretation
Arrhythmias
2.
Ectopic Rhythms – originate outside of the SA node
•
Rapid ectopic rhythms - tachyarrythmias
•
Paroxysmal Supraventricular Tachycardia (sudden)
•
Paroxysmal Atrial Tachycardia (PAT)
Paroxysmal Junctional Tachycardia (PJT)
Paroxysmal Ventricular Tachycardia (PVT)
•
Looks like rapid PVCs? It is
ECG Interpretation
Arrhythmias
2.
Ectopic Rhythms – originate outside of the SA node
•
Atrial Flutter – sawtooth pattern
•
Atrial Fibrillation – sine wave pattern
•
MultiFocal Atrial Tachycardia – (rate?)
Note: if a normal rate, then called a wandering pacemaker.
Wandering Pacemaker
ECG Interpretation
Arrhythmias
•
•
Non-sustained abnormal beats
Premature
•
•
Early – ectopic
Focus discharging spontaneously
Premature atrial contraction (PAC)
o
o
o
o
Can occur on T wave
May be nonconducted
P wave may be missing
Check P-P interval & R-R interval for early wave
ECG Interpretation
Arrhythmias
•
•
Non-sustained abnormal beats
Premature
Premature junctional contraction (PJC)
o
o
o
Retrograde P waves
P waves during or after QRS
P wave may be missing
Junctional Rhythm
ECG Interpretation
Arrhythmias
Premature Ventricular Contraction (PVC)
Early-before atria (P) can begin a new cycle
Enormous QRS – deflections are tall, deep, wide
Compensatory pause – refractory period
Unifocal or multifocal
Coupled with normal beats
Bigeminy - every 2nd beat
Trigeminy – every 3rd beat
Quadrigeminy – every 4th beat
ECG Interpretation
Arrhythmias
Premature Ventricular Contraction (PVC)
Sign of parasystole – 2 pacemakers – sinus & vent.
Runs of PVCs- sign of impending danger
6 PVC’s in one minute – warning of possible V tach.
3 or more in a row are technically considered V tach.
ECG Interpretation
Arrhythmias
Premature Ventricular Contraction (PVC)
PVC on T
PVC falling on T is much too early
Vulnerable period
Called R on T phenomenon
Can lead to V tach
Ventricular Tachycardia
Ventricular Fibrillation
R on T phenomenon
Practice Rhythms
Practice Rhythm
V tach
Rate: __________
Rhythm: _____
P-R interval: ____
QRS: ________
P wave: _______
Rhythm interpretation: ____________________________
Practice Rhythm
V tach
Rate: 300
Rhythm: reg./rapid
P-R interval: n/a
QRS: wide
P wave: none
Rhythm interpretation: Vetricular Tachycardia
Practice Rhythms
Rate: __________
Rhythm: _____
P-R interval: ____
QRS: ________
P wave: _______
Rhythm interpretation: ____________________________
Practice Rhythms
Rate: 75
Rhythm: regular
P-R interval: .16
QRS: .08
Rhythm interpretation: normal sinus rhythm
P wave: present norm.
Practice Rhythms
Sinus Bradycardia
Rate: __________
Rhythm: _____
P-R interval: ____
QRS: ________
P wave: _______
Rhythm interpretation: ____________________________
Practice Rhythms
Sinus Bradycardia
Rate: 43
Rhythm: reg./slow
P-R interval: .20
QRS: .08
Rhythm interpretation: sinus bradycardia
P wave: pres. reg.
Practice Rhythms
Rate: __________
Rhythm: _____
P-R interval: ____
QRS: ________
P wave: _______
Rhythm interpretation: ____________________________
Practice Rhythms
Rate: 125
Rhythm: reg./rapid
P-R interval: .16
QRS: .08
Rhythm interpretation: sinus tachycardia
P wave: pres.
Atrial Flutter
Rate: __________
Rhythm: _____
P-R interval: ____
QRS: ________
P wave: _______
Rhythm interpretation: ____________________________
V tach
Rate: __________
Rhythm: _____
P-R interval: ____
QRS: ________
P wave: _______
Rhythm interpretation: ____________________________
V tach
Rate: 300
Rhythm: reg./rapid
P-R interval: n/a
QRS: wide
Rhythm interpretation: v - tach
P wave: n/a
Practice Rhythms
A fib answer
Rate: __________
Rhythm: _____
P-R interval: ____
QRS: ________
P wave: _______
Rhythm interpretation: _____________________
Practice Rhythms
A fib answer
Rate: A – 350+
Rhythm: irreg./rapid P wave: irreg.
P-R interval: n/a
QRS: none
Rhythm interpretation: atrial fibrillation
Atrial flutter
Rate: __________
Rhythm: _____
P-R interval: ____
QRS: ________
P wave: _______
Rhythm interpretation: ____________________________
Atrial Flutter
Rate: a-350/v-125
Rhythm: irreg./rapid P wave: rapid
P-R interval: n/a
QRS: .08
Rhythm interpretation: atrial flutter
Junctional Escape Beat-Rhythm
Rate: __________
Rhythm: _____
P-R interval: ____
QRS: ________
P wave: _______
Rhythm interpretation: ____________________________
Junctional Escape Beat-Rhythm
Rate: a-0/v- 35-60
Rhythm: irreg./slow P wave: none
P-R interval: n/a
QRS: .08
Rhythm interpretation: junctional escape rhythm
PVC
Rate: __________
Rhythm: _____
P-R interval: ____
QRS: ________
P wave: _______
Rhythm interpretation: ____________________________
PVC
Rate: 73
Rhythm: irreg.
P-R interval: .12
QRS: 1 wide (.16)
P wave: 1 missing
Rhythm interpretation: PVC (premature ventricular contraction)
Sinus Arrest
~ 3.5 sec
between
beats
Triplet – run of pvc
Triplet – run of pvc
Trigeminy
Trigeminy
Quadrigeminy
Quadrigeminy
ECG Interpretation
Arrhythmias
3.
Conduction Blocks – electrical blocks which slow
or prevent passage of electrical stimuli
(depolarization)
•
Sinus Block – “sick sinus”
•
•
•
•
Unhealthy SA node may skip or fail for at least one cycle
No P or QRST – “lost beat”
Resumes pacing as if nothing happened
Same P waves before and after
ECG Interpretation
Arrhythmias
Conduction Blocks
3.
•
Atrioventricular Blocks (AV Blocks)
First Degree AV Block
Prolonged P-R interval (> .20 sec) every cycle
Consistent P-R - prolonged by same amount
First Degree Block
ECG Interpretation
Arrhythmias
Conduction Blocks
3.
•
Atrioventricular Blocks (AV Blocks)
Second Degree AV Block – 2 types
o
Mobitz Type I (Wenckebach)
o P-R interval progressively increases until a P is not
followed by a QRS.
o Last P stands alone
o Always a P but eventually no QRS
Second Degree – Mobitz I- Wenckebach
ECG Interpretation
Arrhythmias
Conduction Blocks
3.
•
Atrioventricular Blocks (AV Blocks)
Second Degree AV Block
o
Mobitz Type II
o QRS is “dropped” - not conducted even though P wave is
present and normal
o All is normal before and after event
o No lengthening P-R (no warning)
o Sign of more serious conduction problems
Second Degree Mobitz II -
ECG Interpretation
Arrhythmias
Conduction Blocks
3.
•
Atrioventricular Blocks (AV Blocks)
Third Degree AV Block
Total lack of conduction through the AV node
Rate and the interval between the QRS depend upon the origin
of the escape mechanism.
May progress to ventricular standstill
Independent P waves and QRS's with no relationship between
the two (AV dissociation – separate atrial & ventricular rates)
Third Degree Block
AV Block
ECG Interpretation
Arrhythmias
4.
Preexcitation Syndromes
Key Feature: Short PR interval
Wolf-Parkinson-White
(also note wide QRS with delta wave in some leads)
Lown-Ganong-Levine
(normal QRS; no delta wave)
ECG Interpretation
12-Lead ECG
Analysis:
1.
2.
3.
4.
5.
6.
7.
8.
Determine regularity of rhythm.
Determine rate.
Examine P waves
Measure PR interval
QRS complex width
Determine Axis
Examine ST Segment (normal is < 1mm elevation or
depression)
Check T waves (upright except for aVR, V1, and
possibly III)
ECG Interpretation
12-Lead ECG
Common Abnormalities:
Axis Deviation
1.
•
Normal axis between 0° and 90°
Bundle Branch Blocks
2.
•
Right Bundle Branch Block
•
Wide QRS
RSR’ in V1 and V2; T wave inversion and ST depression
Deep S wave in V5/V6
Left Bundle Branch Block
Wide QRS
Broad R waves in V5/V6; T wave inversion and ST depression
Deep S waves in V1/V2
ECG Interpretation
12-Lead ECG
Hemiblocks
3.
•
•
Anterior: left axis deviation
Posterior: right axis deviation
Bifascicular block: right bundle branch block +
hemiblock
5. Ventricular Hypertrophy
• Right: R S in V1
• Left: R in V1 + S in V5 35 mm
• T wave inversion and ST depression in both.
4.
ECG Interpretation
12-Lead ECG
Ischemia
• ST depression; inverted symmetrical T waves
7. Infarct
• Acute (also referred to as injury)
ST elevation
• Old
Abnormal Q waves (.04 sec wide; 1/3
height of R wave)
6.
Diagnostic Value of ECGs
(Chapter 6, pages 124-129 in text)
Sensitivity: percentage of people with CAD who
have a positive ECG test.
70% for ECG stress test
TP/(TP + FN)
Specificity: percentage of people without CAD
who have a negative ECG test.
77% for ECG stress test
TN/(TN + FP)
Questions?
End Electrocardiography