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The effect of TLR on transplantation and tolerance of the cornea

Mee Kum Kim M.D

1 , Jinho Jeong M.D

1 , Won Ryang Wee M.D

1 , Jin Hak Lee M.D

2

1 Department of ophthalmology, Seoul National University Hospital, Seoul, Korea 2 Department of ophthalmology, Seoul National University Bundang Hospital, Bundang, Korea

The authors have no financial interest in the subject matter of this poster.

Purpose

To investigate

the role of toll-like receptor (TLR)

 On the

MHC mismatched allograft rejection in corneal transplantation

 On the

immune tolerance of the cornea

Materials and Methods

Mouse cell culture

 Group  Group K : keratocytes only    Group D : dendritic cells only (DC2.4*) Group M : macrophages only (Raw264.7*) Group K+D : keratocytes + dendritic cells  Group K+M : keratocytes + macrophages : 1x10 4 cells : 1x10 4 cells : 1x10 4 cells : 0.5x10

4 + 0.5x10

4 cells : 0.5x10

4 + 0.5x10

4 cells

Mouse primary corneal keratocyte DMEM/F12 (1:1) with 10% FBS and 1%PS Mouse DC cell line: DC2.4

*

RPMI1640 with 10%FBS and 1%PS

Mouse macrophage cell line: Raw264.7*

DMEM with 10%FBS and 1%PS

* Kindly provided by Kim TJ

 TLR stimulation with agonist  LPS (TLR4 agonist)  Pam3csk4 (TLR2 agonist)  Change in TLR expression and its products of TLR signaling pathway 

TLR2 and TLR4

expression  RT-PCR  Stimulation for 6 and 24 hours with LPS or Pam3csk4

TLR

Phospho-IkappaB-alpha (p-I κB-α)

 Western blot  Stimulation for 2, 6, and 24 hours with LPS or Pam3csk4  Cytokine assay    ELISA Candidate cytokines :

IL-6, TNF α, TGF-ß, IL-10

Stimulation for 24 hours with various concentration   LPS : 1, 10, 100, 1000, 10000 ng/ml Pam3csk4 : 1, 5, 10, 50, 100 ng/ml

Corneal transplantation

 Group (Donor     C3H  Recipeint) C57BL/6 (B6)  B6 C3H/HeN (C3H)  B6 TLR4 knockout (TLR4KO) : Autograft (n=10) : MHC mismatched allograft (n=11) : (n=13)

TLR4 knock-out mice on a C57BL6 (B6 background) were bred at a SPF animal facility and were kindly provided by Dr. S-Y Seong at the Department of Microbiology and Immunology, Seoul National University College of Medicine

   Transplantation procedure   Donor /recipient : 3.0 / 2.0 mm trephine 8 to 10 interrupted 10-0 Nylon sutures Clinical evaluation  Analysis of graft survival using Slit-lamp biomicroscopy   Definition of rejection (end point) : 100% graft edema Kaplan-Meier survival analysis (Log rank (Mantel-Cox) test) Immune cell assay  Cervical lymph node and spleen   12 days & 4 weeks after transplantation Flow cytometry (FACSCalibur) and FlowJo software  CD11c, CD80/CD86: CD4/8, CD44/69: CD4/8, IFN 

Results

TLR expression after agonist stimulation

TLR4 in M, D, and K

TLR2 in D and K

P-I κB-α expression after agonist stimulation

P-IκB-α in M, D, and K

P-IκB-α in M,and D

Cytokine secretion after agonist stimulation

After LPS stimulation

Coculture : K + D Markedly increased anti-inflammatory cytokine IL-10 and TGF-β1 in coculture condition

Coculture : K + M

K : B6 Keratocyte D : Mouse dendritic cell line (DC 2.4) M : Mouse macrophage cell line (Raw 264.7

)

After Pam3csk4 stimulation

Coculture : K + D Markedly increased anti-inflammatory cytokine IL-10 and TGF-β1 in coculture condition

Coculture : K + M

K : B6 Keratocyte D : Mouse dendritic cell line (DC 2.4) M : Mouse macrophage cell line (Raw 264.7

)

Corneal allograft survival and immunologic profiling

 Median survival time :

C3H

B6 > C3H

TLR4KO P<0.05, Log Rank test

 IFN γ secreting CD8 T cell :

C3H

B6 < C3H

TLR4KO

0,7 0,6 0,5 0,4 0,3

CD8 IFN-

in L/N at 12 days

0,2 0,1 0 B6-B6

P<0.05, Mann Whitney U test

CH3-B6 CH3-TLR4KO B6

Lymph nodes CD86 1.90

At 12 days 1.81

3.53

2.84

CD 11c 1.15

At 4 weeks 5.49

2.70

10.64

CD 11c CD80 Spleens CD80 CD86 2.27

3.48

1.57

CD 11c 4.13

2.77

3.17

1.75

CD 11c 1.95

C3H

B6 C3H

TLR4KO C3H

B6 C3H

TLR4KO

0.27

0.31

0.74

0.29

0.29

0.60

0.34

0.52

0.93

C3H

B6 C3H

B6 C3H

TLR4KO

Conclusions

 TLR4

is likely to be involved in immune modulation of MHC mismatched corneal allograft rejection

Fibroblast seems to be involved in immune modulation through TLR signaling