The_Child_with_a_Gastrointestinal_Alteration - Lake
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Transcript The_Child_with_a_Gastrointestinal_Alteration - Lake
Pilar C. Smith, RN, BSN
Lake Sumter Community College
Cleft Lip and Palate
Cleft lip results when
the medial nasal and
maxillary processes fail
to join at 6 to 8 weeks of
gestation.
Cleft palate results from
failure of the primary
patatal shelves, or
processes, to fuse at 7 to
12 weeks of gestation.
Manifestations/Diagnostic Assessment
Cleft lip:
A notched vermilion border, cleft that involve the
alveolar ridge and dental anomalies.
Cleft palate:
includes nasal distortion, midline or bilateral cleft with
variable extension from the uvula, soft and hard palates
and exposed nasal cavity. First sign may be formula
coming from the nose
Diagnostic is based on observation at birth and
complete examination in the neonatal period.
Assessment
Assess infant’s ability to suck, swallow and breathe
without distress and handle normal secretions.
Assess and record parents’ reactions as well as
interaction with the neonate.
Provide information about the causes of the defect
Encourage bonding through touching, holding
and examining their newborn.
Point out the newborn’s positive attributes.
Intervention
Describe the degree of cleft and impairment of sucking.
Modify feeding techniques as needed to allow adequate growth
Provide alternative assistive feeding devices
Hold infant in a more up-right position
Burp infant more frequently
Write down feeding program for parents to use at home
Provide emotional support and positive reinforcement to parents
Keep and accurate record of child’s growth by using a growth
chart.
Orthopedic Latham device is use to expand and realign
Explain preoperative and postoperative procedure
Surgical Intervention
Cleft lip repair is usually performed by age 3 to 6
months. Cosmetic modification may be needed at
age 4 to 5 years.
Cleft lip repair is usually performed by age 3 to 6
months. Cosmetic modification may be needed at
age 4 to 5 years.
Surgical Intervention
Preoperative care:
Oral feeding withheld for 6 hours
IV line placement
Postoperative care:
Keep straws, pacifiers, spoons, or fingers away from the
child’s mouth for 7 to 10 days
Do not take temperature orally
Advance the child’s diet as tolerated from clear liquids to
soft diet within 48 hours.
Nursing Diagnoses
Alteration in nutrition less then body requirement
related to the inability to suck
The child will drink the recommended amount of
feeding within 30 minutes
Knowledge deficit about feeding techniques and
surgery related to unfamiliarity with the information
The parent will understand expected preoperative and
postoperative feeding techniques.
Esophageal Atresia with
Tracheoesophageal Fistula
Esophageal atresia with TEF are congenital
malformations in which the esophagus terminates
before it reaches the stomach and/or a fistula is
present that forms an unnatural connection with the
trachea.
Manifestations
Failure to pass suction catheter or NGT at birth
Excessive oral secretions
Vomiting
Abnormal distention
Airless, scaphoid abdomen (atresia without
fistula)
3 “Cs” coughing and choking with feeding and
cyanosis
Diagnostic Assessment:
Hx. of maternal polyhydramnios (prenatal
clue)
IF NG tube cannot be passed 10 to 11 cm
beyond the gum line
Abdominal radiograph (X-ray) with watersoluble contrast medium < 1ml via NGT
Bronchoscopy and endoscopy
Management
Keep infant warm and oxygenated
Keep infant supine with the HOB elevated to keep
gastric secretions from entering the lungs
NGT aspirate every 5 to 10 minutes to keep the keep
the proximal pouch clear
Intravenous IV fluids are essential
Surgical repair: Ligation of the fistula and end-to-side
anastomosis of the atresia
Nursing diagnosis and planning(read pp 520-522)
GER in children
GER vs GERD.. Page 522
Symptoms range from physiologic to pathologic
Diagnostic exam includes barium swallow, upper GII
study, endoscopy
Management: dietary alterations, positional changes,
medications and surgery
Diet: predigested formulas.. Nutramigen or
progestimal, freq, small feedings, freq. burping, ?
Thickened feedings
Positioning : supine
GER continued
Medications: antacids for symptom relief, H2 receptor
antagonists.. Cimetidine, ranitidine to decrease acid
secretion, mucosal protectors, eg. Sucralfate for barrier
protection and PPI’s ompeprazole to suppress gastric
acid secretion and prokinetic agents metoclopramide
to accelerate gastric emptying
Prevent complications
Nursing care plan on page 527-529
Constipation and Encopresis
Definition: page 529
Etiology: change in diet, lack of exercise, emotional
stress certain drugs pain from anal fissure or excessive
milk intake
Encopresis generally affects a child from 3 to 7 years
old more often in boys than girls.
Constipation affects children at any age.
s/s: constipation versus encopresis page 530
Abdominal x-ray DRE
Constipation and Encopresis cont..
Therapeutic Management Page 530
Nursing Care:
Assessment
Nursing Diagnosis and Planning..page 531
Interventions: Overcoming withholding, dietary
changes, changing the retention habit, emotional
support, home care
IBS
Increased intestinal motility which can lead to spasm
and pain
Etiology: stress and emotional factors, lactose
deficiency, tends to occur in families with a HX of
bowel disturbances can occur as constipation or
diarrhea
S/S : abd pain unrelated to meals or activity Normal
growth mucous in stool
IBS cont
Do definitive treatment Goal is aimed at identifying
and reducing triggers and reducing bowel spasms
which decreases triggers
Diet is healthy well balanced moderate fiber lower fat
diet
Encourage child to eat slow and no carbonated
beverages, no juices
Med’s: EC peppermint oil capsules, antispasmodics
such as Levsin and Donnatal and antidepressants in
severs cases.
Psychosocial support
Gastroenteritis
Cause: viruses, bacteria and parasites
Etiology: ingestion of contaminated food or water and
person to person contamination. High risk groups are
children in daycare centers, preschools and long term
care facilities and those infected with HIV
Giardia is the most common seen in children in day
care and rotovirus is the most common seen in infants
and young children
Page 535 characteristics of infectious gastroenteritis
Gastro. Cont.
S/S diarrhea, vomiting abdominal pain tenesmus and
fever dehydration is a consequence of gastro
Stool culture O&P
Management: replace water and electrolytes
Prevention Rotovirus vaccine at age 2,4, 6 months
What do we assess?
Nursing Diagnosis- page 537
Interventions- page 537
Ulcers
Types
Etiology
Diagnostic tests
S/S
therapeutic management
Appendicitis
Pain progressing in intensity and localizing to the RLQ
at Mc Burney point page 538
Diagnostic evaluation?
Therapeutic management?
Nursing Diagnosis
Interventions
Volvulus
Malrotation or twisting of the bowel that results in
obstruction.
S/S pain, bilious vomiting
Surgery is the cure
Hypertrophic Pyloric Stenosis
Hypertrophic pyloric stenosis results when the circular
area of muscle surrounding the pylorus hypertrophies
and obstructs gastric emptying.
Manifestation
Progressive projectile vomiting , especially after eating
On palpation:
Right upper quadrant movable firm olive-shaped mass can be
felt
Visible peristaltic waves
from left upper to right upper quadrant immediately before
vomiting.
Dehydration:
absence of tears, a weak cry, depressed fontanel, poor skin
turgor and dry mucous membrane.
Metabolic Alkalosis
Diagnostic Assessment
history of vomiting, visible peristalsic waves and a
palpable pyloric mass
X-ray or US of the abdomen,
Barium swallow
Metabolic alkalosis
Decreased serum potassium and sodium levels
Increased Ph and sodiumbicarbonate, and decreased
chloride.
Indirect Bilirubin may be elevated.
Management
IV fluids and electrolyte replacement
NGT for stomach decompression
Surgery:
Pyloromyotomy, an incision of the pyloric
muscle to release the obstruction done by
Laparoscopy.
Pre and postoperative care and home care.(read
Rowen pp.546-547)
Assess family dynamics and support system
Nursing Diagnoses
Fluid and electrolyte deficit related to vomiting
Alteration in nutrition less then body requirement
related to vomiting
Intussusception
Inussusception is an invagination of a section of the
intestine into the distal bowel that causes bowel
obstruction
Manifestation
Classic sign
Passage of bloody mucus (“currant jelly”) stool and diarrhea
A sausage- shape abdominal mass
Listlessness
Paroxysm pain (pain that comes and go) than becomes
more constant
Distention, hypo or hyperactive bowel sounds
Sign and symptoms of dehydration
Symptoms of shock and sepsis if obstruction has been
present for longer than 12 to 24 hours
Fever, increased heart rate, changes in LOC
Management
The goal is to restore the bowel to its normal
position and function.
Hydrostatic reduction if child does not have
symptoms of shock or sepsis.
Barium enema or air enema.
Ultrasound-guided isotonic saline enemas
Surgery:
Laparoscopy is use if no bowel necrosis is present.
Nursing Diagnoses
Ineffective tissue perfusion related to (GI) bowel
compression.
Acute pain related to bowel obstruction and surgery
Hirschsprung disease
Hirschsprung disease is
caused when
parasympathetic ganglion
cells in the wall of the large
intestine(colon) do not
develop before birth.
Without these nerves, the
affected segment of the
colon lacks the ability to
relax and move bowel
contents along.
Manifestation
Delayed or absence of meconium stool in the neonatal
period (cardinal sign)
Constipation since neonatal period
Frequent passage of foul-smelling ribbonlike or
pelletlike stools
Malnutrition
Abdominal pain and distention
Failure to thrive
Diagnostic Assessment
Rectal examination:
tight internal sphincter and absence of stool, follow by
an explosive release of gas and stool
Barium enema
Rectal biopsy (definitive)
Absence of ganglia cells confirm the diagnosis
Management
Treatment for mild to moderate
Stool softener to relieve constipation and rectal
irritation
Treatment for moderate to severe:
Surgical intervention: the aganglionic portion of the
intestine is removed in two steps.
In the neonatal period, performing a temporary colostomy.
A complete surgical repair when the child weights 8 to 10 kg
(18 to 20 Lbs), the colostomy is closed during this time.
Assess family member’s concerns and way of coping with
the problem
Nursing Diagnoses
Constipation related to
aganglionic bowel and
inadequate peristalsis
Impaired skin integrity
related to colostomy
and surgical repair.
Celiac disease
Celiac disease, also known as gluten enteropathy
results from the inability to digest fully the protein
part of wheat, barley, rye, and oats.
This results in the accumulation of glutamine in the
intestine, which has a toxic effect in the mucosal cell.
This leads to atrophy of the villi and decrease
absorption of fat, carbohydrates and vitamins.
It can cause severe diarrhea and dehydration.
Manifestation
Profuse diarrhea especially foul-smelling fatty stools
Growth failure: bellow the twenty-fifth percentile
Abdominal distention and ascites
Anemia
irritability
anorexia
muscle wasting
folate deficiency
Edematous extremities
Diagnostic Assessment
Serum antigliadin antibody (AGA) assay
allows continued assessment and evaluation of dietary
changes
Tissue transglutaminase (tTG)
Breath hydrogen excretion test
identify carbohydrate malabsorption.
Management
All wheat, rye, barley, and oat should be eliminated
from the diet
Replaced with corn and rice
Fat-soluble vitamins supplement and folate
Fluid replacement if child is dehydrated from severe
watery diarrhea
Correct acid-base imbalance
Albumin to treat shock
Corticosteroids to decrease severe mucosal
inflammation
Nursing Diagnoses
Imbalance nutrition less then body requirement
related to malabsorption
Delayed growth and development related to
malnutrition
Lactose Intolerance
Inability to tolerate lactose
Most case of lactose intolerance are the result of
inadequate lactase
S/S diarrhea that is frothy, abdominal distention,
cramping pain and increase flatus
+1 or greater sugar values on stool clinitest can support
the diagnosis
Management: remove the milk
ND: page 553
reference
Health topics A-Z
http://adam.about.com/encyclopedia/Pyloric-stenosis.htm
University of Virginia Health System
http://www.healthsystem.virginia.edu/uvahealth/peds_digest/intussus.cfm
Healthline connect to better health
http://www.healthline.com/channel/hirschsprung-disease_images
Rowen,S. J., Weiler, J. A. (2007). The child with a gastrointestinal alteration. In
NursingCare of children principles and practice. (pp 507-5596). St. Louis,
Missouri: Saunders