Constrictive Cardiomyopathy Versus Restrictive Cardiomyopathy Echocardiography Dr Djilali Hanzal Cardiologist National Guard Hospital Outline  Background  Physiology  Clinical Features  Echocardiography :       M mode 2D Doppler Tissue Doppler Strain Imaging Conclusion.

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Transcript Constrictive Cardiomyopathy Versus Restrictive Cardiomyopathy Echocardiography Dr Djilali Hanzal Cardiologist National Guard Hospital Outline  Background  Physiology  Clinical Features  Echocardiography :       M mode 2D Doppler Tissue Doppler Strain Imaging Conclusion.

Slide 1

Constrictive Cardiomyopathy
Versus
Restrictive Cardiomyopathy
Echocardiography

Dr Djilali Hanzal
Cardiologist
National Guard Hospital


Slide 2

Outline
 Background

 Physiology
 Clinical Features
 Echocardiography :








M mode
2D
Doppler
Tissue Doppler
Strain Imaging
Conclusion


Slide 3

Etiology CP

Bertog SC, J Am Coll Cardiol. 2004;43(8):1445.


Slide 4

Symptoms

Tajik AJ Circulation. 1999;100(13):1380.


Slide 5

Varieties of constrictive pericarditis

Rien muller et al .J Thorac Imaging 1993


Slide 6

J Am Coll Cardiol 2004;43;1445-52


Slide 7

Anatomy
Lt. Atrium is not
Completely
intrapericardial

All other cardiac chambers are
completely intrapericardial

Pulmonary Veins are
completely intrathoracic


Slide 8

Effect of Inspiration
Normal Pericardium
Intra thoracic pressure
Venous return

Constrictive Pericarditis
 Intra thoracic pressure
 Venous return

Transient size of RV

 RV not expanded

Normal LV filling

 Abnormal LV filling

Uptodate 2011


Slide 9

Mechanism

• FILLING

IMPAIREMENT
• LV-RV

INTERDEPENDANCE


Slide 10

Physiology
CP vs RCM
Constrictive Pericarditis
Myocardial compliance is NL
Pericardium not compliant
Septum compliant
Rapid early diastolic filling
cardiac volume is fixed by the
pericardium

Respiratory effect of LV
on the RV

Restrictive
Ab-Nl Myocardial compliance
Pericardium compliant
Septum not compliant
Impedence to filling increases
throughout the diastole

No Respiratory effect of
RV and the LV


Slide 11

Restrictive
Cardiomyopathy
(Myocardial Disorders)

Myocardial
disease

Endomyocardial
disease

Storage disease

Endomyocardial
fibrosis

Infiltrative

Noninfiltrative

Amyloidosis
Sarcoidosis

Idiopathic CMP
Diabetic CMP

Hemochromatosis

E William Hancok, Heart 2001, 86 343-349


Slide 12

Why is it important to make the distinction
RCM vs CP?
 Associated with significant morbidity and

mortality
 Restriction rarely treatable/curable
 Constriction may be curable with surgery.


Slide 13

Inexplained CHF
CXray: No Cardiomegaly ( Clinically, Cxray, BNP..)

Echo: Normal LV systolic
function

Trans mitral Doppler:
Restrictive Pattern: E/A>2

TDI:
(E’>8cm/s, E/E’<15
Normal S wave)

CP

TDI:
E’<8cm/s,E/E’>15

CP

RCM

Cho YH and Schaff.Heart Fail Rev 2012


Slide 14

Inexplained CHF
CXray: No Cardiomegaly ( Clinically, Cxray, BNP..)

Echo: M-Mode, 2-D
Normal LV Systolic Function


Slide 15

M-mode and 2-D
CP
 Pericardial thickening and calcification
 Septal bounce
 Dilated not collapsing Inferior Vena Cava

 Flattening of LV post wall
 Early pathological outward and inward

movement of the IVS
 Color M-mode Propagation


Slide 16

18% of PC had normal thickness


Slide 17

CP
 Differential Dx:







Constrictive Pericarditis
Pericardial Tamponade
Pulmonary Hypertension
LBBB
Right Ventricular Pacing

.

 Paradoxal motion of the IVS

occurring in early diastole


Sensibility 62%,Specificity 93%

Journal of Thoracic Imaging. 27(1):w1, January 2012.


Slide 18


Slide 19

M-Mode CP



Signs reflecting increased
ventricular interdependence
Abrupt early diastolic anterior
motion of the IVS followed by a
rebound toward the LV post wall.

Mastouri et al. Expert Rev Cardiovasc 2010
.


Slide 20

M-Mode CP
 Signs reflecting rapid early



ventricular diastolic filling:
Flattening at the LV post wall

 Sensitivity 92%, Specificity 100%

Voelkel et al ,Circulation. 1978 Nov;58(5):871-5.


Slide 21

M-Mode CP
 Signs reflecting

increased Right Ventr
diastolic pressure
above Pulmonary Art
pressure
• Premature opening of the
pulmonary valve



Sensibility 14%,Specificity 100%

Mastouri et al. Expert Rev Cardiovasc 2010


Slide 22

Sensibility 74%,Specificity 91%
Am J 2001,87,86-94


Slide 23

RCM 2-D
 Small LV cavity with

large atria
 Increased wall
thickness ( especially
in interatrial septum in
Amyloidosis)
 Thickened valves and
granular sparkling
texture (amyloidosis)


Slide 24

Inexplained CHF
CXray: No Cardiomegaly ( Clinically, Cxray, BNP..)

Echo: M-Mode, 2-D
Normal LV Systolic Function

Echo-Doppler:
Restrictive Pattern:
E/A>2,DT<150ms,IVRT<60ms
AV Inflow


Slide 25

Echo-Doppler
 Mitral and Tricuspid Inflow
 IVRT
 TR

 Hepatic Veins
 Pulmonary Regurgitation
 Pulmonary Veins
 Superior Vena Cava


Slide 26

CP

Specificity
67%,
Sensibility
86%

JACC,1994 Jan;23(1):154-62
J Am Coll Cardio 1994 jan.23,154-


Slide 27

Constriction: Non-respirophasic
Mixed Restriction and Constriction
Marked increase in Preload
• Provocation test with head-up tilting or
sitting position with decrease of the
preload may unmask the CP.

Maisch, Seferovic, Ristic et al.ESC guidelines on pericardial
disease, E J 2004


Slide 28


Slide 29

AF and CP


Slide 30

AF and CP

J Am Coll Cardio 2001;37:1936-42


Slide 31

CP

JACC 1994 Jan;23(1):154-62


Slide 32

Diagrammatic representation of the transmitral early (E-wave) and late (A-wave) velocities
during diastole throughout the respiratory cycle.

Nihoyannopoulos P , Dawson D Eur J Echocardiogr
2009;10:iii23-iii33
Published on behalf of the European Society of Cardiology. All rights reserved. © The Author
2009. For permissions please email: [email protected]


Slide 33

CP


Slide 34

CP


Slide 35

Normal

CP

Specificity79%,
Sensitivity 86%
Circulation 2002, Rajagopalan et al. AJC 2001


Slide 36

CP


Slide 37

Normal

CP

RCM

PV is Respirophasic
PV is not Respirophasic


Slide 38

CP


Slide 39

CP vs COPD
CP


Slide 40

Inexplained CHF
CXray: No Cardiomegaly ( Clinically, Cxray, BNP..)

Echo: Normal LV systolic
function

Echo-Doppler:
Restrictive Pattern:
E/A>2,DT<150ms,IVRT<60ms
AV Inflow

Tissue Doppler:
Annular TDI


Slide 41

Specificity 89%,Sensibility100%

Rajagopalan et al
.Am.J.Cardio 2001


Slide 42

E/e’=6

Am J Cardiol 2004;93:886-890


Slide 43

MITRAL “ANNULUS REVERSUS”
Normal
E’ Lateral > E’ Septal

CP
E’ Lateral< E’Septal

RCM
E’ Lateral =E’ Septal

Reuss et al.Eur J Echocardiography 2009


Slide 44


Slide 45

Inexplained CHF
CXray: No Cardiomegaly ( Clinically, Cxray, BNP..)

Echo: Normal LV systolic
function

Echo-Doppler:
Restrictive Pattern:
E/A>2,DT<150ms,IVRT<60ms
AV inflow

Tissue Doppler:
Annular TDI

Strain Imaging


Slide 46

Myocardial Mechanics in RCM and CP
Deformation
Parameter
Longitudinal
Strain
Circumferential
Strain

CP
Normal

Decreased

JACC Cardiovasc Imaging. 2008 Jan;1(1):29-38

RCM
Decreased

Normal


Slide 47


Slide 48

CP

RCM

J Am Soc Echocardiogr 2009:22:24-33

2-D Speckle-tracking


Slide 49

CP

RCM

Em: Longitudinal early diastolic
lengthening velocity

J Am Soc Echocardiogr 2009:22:24-33


Slide 50


Slide 51


Slide 52

Too much for Diastology


Slide 53

Conclusions
 Dx has important therapeutic implications
 Clinical Presentaion similar
 Echocardiography (Doppler,TDI, Strain/Strain

rate) have increased yield.
 Cardiac catheterisation still considered

mandatory.


Slide 54

End


Slide 55

Inexplained CHF
CXray: No Cardiomegaly ( Clinically, Cxray, BNP..)

Echo: Normal LV systolic
function

Echo-Doppler:
Restrictive Pattern:
E/A>2,DT<150ms,IVRT<60ms
AV inflow

Tissue Doppler
Annular TDI

Hemodynamic

Strain


Slide 56

QTDI

CP

Normal
International J of Cardio
137(2009)22-39


Slide 57

RCM
International J of Cardio 137(2009)22-39


Slide 58

Major historical events in CP

Korean Circ J 2012;42:143-150