Neuropathic pain

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Transcript Neuropathic pain

Neuropathic pain

1. Mechanism 2. Characteristic 3. Diagnosis 4. Treatment

Neuropathic pain

Mechanism of pain: caused by cancer

nerve compression - nerve root compression caused by a collapsed vertebra

total tumor mass = neoplasm + surrounding inflammation

nerve infiltration by cancer

nerve injury

Neuropathic pain

Mechanism of pain: caused by treatment

postoperative (neurotomy)

phantom limb pain, post-mastectomy pain

radiotherapy (fibrosis) e.g. Brachial plexopathy

chemotherapy - peripheral neuropathy (wincristine, cisplatine, taxol)

Neuropathic pain

Mechanism of pain:

post-herpetic neuralgia

diabetic neuropathy

post-stroke pain

uraemic neuropathy

Neuropathic pain

Pain characteristic:

superficial burning pain

spontaneous stabbing/shooting pain

boring and radiating pain

allodynia - pain caused by a stimulus which does not normally provoke pain

hyperalgesia - an increased response to a stimulus which is normally painful

Neuropathic pain

Diagnosis:

history

clinical examination

neurological examination

MRI / CT

Neuropathic pain

Treatment: I. Adiuvant analgesics II. Corticosteroids III. Analgesics (opioids) IV. Neurolysis, spinal analgesia

Neuropathic pain

Corticosteroids (reduces total tumor mass) e.g. Dexamethason 16-24mg at the begining and then reduse dose Antidepressants - tricyclic antidepressants (amitriptyline, desipramine, doxepin, imipramine, clomipramine) SSRI (paroxetine, citalopram, fluoxetine)

Neuropathic pain

Amitriptyline is effective in migraine and other types of headache, chronic low back pain, post-herpetic neuralgia, fibromialgia, painful diabetic polyneuropathy, central pain, cancer pain.

Superficial burning pain, allodynia = tricyclic antidepressants

10-25mg nocte at the begining; max 75mg

relief may not occur for 4-5 days, for effect you have to wait even 1-2 weeks

Neuropathic pain

Anticonvulsants - carbamazepine, gabapentin, valproate, oxcarbazepine, lamotrigine

spontaneous stabbing/shooting pain

carbamazepine 200-1600mg; effect after 10-14 days

adverse effects!

gabapentin - 300-3600mg; effect after one week

Neuropathic pain

Other drugs:

oral local anasthetics lignocaine infusions - mexiletine 450-600mg ;

    

NMDA receptor antagonists - dextromethorphan, ketamine (in subanaesthetic doses), bupivacaine, methadon muscle relaxants - Baclofen 10-15mg >>75-100mg topical agents EMLA - capsaicin, lignocaine patch, benzodiazepines and neuroleptics spinal analgesia - epidural and intrathecal routes.

A 4-step analgesic ladder used either alone or in conjunction with the WHO 3-step ladder

Tricyclic antidepressant

or

anticonvulsant

Step 1

Tricyclic antidepresant

and

anticonvulsant

Step 2

Class I antiarrhytmic

or

cetamine

Step 3

Spinal analgesia

Step 4

Bone pain

1. Mechanism 2. Pain characteristic 3. Diagnosis 4. Treatment

Bone pain

Mechanism :

metastases - breast, prostate, thyroid, kidney, lung, colon

cancer infiltration of the bone

pathologic fracture

Bone pain

Pain characteristic: - continuous, aching and localized pain - is exacerbated by movements and sneezing - may be unifocal multifocal generalized

Bone metastases

Symptoms:

pain (75%)

neurological symptoms

pathologic fracture

hypercalcaemia

bone marrow failure

Bone pain

Diagnosis:

history

clinical examination

rtg

scintigram

MRI / CT

Bone pain

Treatment:

surgery - bone stabilisation, tumor excision

radiation therapy - is usually considered when bone pain is focal and poorly controlled with an opioid

chemotherapy (chemosensitive tumors)

hormonotherapy (hormonosensitive tumors breast, prostate)

Bone pain

Radiopharmaceuticals high bone turnover that are absorbed at areas of strontium-89, rhenium-186, samarium-153

strontium is only potentially effective in treatment of pain due to osteoblastic bone lessions or lession with an osteoblastic component e.g. prostate cancer metastases

strontium - initial clinical response occurs in 7-21 days - the usual duration of benefit is 3-6 months

Bone pain

Non-steroidal anti-inflammatory drugs (NSAID)

opioids

corticosteroids

bisphosphonates (clodronate, pamidronate)

calcitonin

neurolysis, spinal analgesia

Bone pain

Bisphosphonates

- inhibit osteoclast activity and reduce bone resorption -provide analgesia and decrease the use of analgesics

clodronate:

- intravenous dose 600mg weekly - oral dose - 1600mg daily

pamidronate:

- intravenous dose 60-90mg every 3-4 weeks - is safe in patients with impaired renal function - adverse effect: occasional hypocalcaemia, nausea

Bone pain

Calcitonin: mechanism of action is unclear - increase endorphin levels in the central nervous system - interact with the serotonergic system - anti-inflammatory action - direct effect on osteoclasts

calcitonin - subcutaneous - relatively low dose at the begining, then gradually increased to 200 IU - intranasal- 200 IU in one nostril; alternating nostril everyday

Spinal cord compression

Neurological emergency

3-5% of patients with advanced cancer

40% is associated with cancers of the breast, lung, prostate

others are associated with: renal cell cancer, lymphoma, myeloma, melanoma, sarcoma, colorectal cancer

very rarely spinal cord syndromes are due to epidural or cord metastases

Spinal cord compression

Mechanism of compression: - metastatic spread to vertebral body or pedicle - 85% - tumor extension through intervertebral foramina - 10% - intramedullary primary - 4% - haematogenous dissemination - epidural space - 1%

Spinal cord compression

Clinical presentation: pain (>90%) - pain of long duration which suddenly changes -pain is aggravated by lying down - pain may occur spontaneously - radicular pains are often exacerbated by neck flexion or straight leg raising, by coughing, sneezing or straining - funicular pain is less sharp, has a more diffuse distribution and is sometimes described as a cold unpleasant sensation

Spinal cord compression

Clinical presentation: - weakness > 75% - paraesthesiae - sensory loss (>50%) starting in the feet and moving proximally (is helpful in defining the level of the compression) - sphincter dysfunction >40% loss of sphincter function is a bad prognostic sign

Spinal cord compression

Diagnosis: - history - clinical examination - neurological examination - rtg - shows vertebral metastasis / collapse - MRI is the investigation of choice - CT with myelography may be helpful if MRI is not available

Spinal cord compression

Treatment: - high-dose steroids and radiation should be offered to all patients.

Steroids can reduce pain and preserve neurological function; initial dosage - 100mg i.v.bolus (usually 24 50mg) followed orally halving of the dose every third day until the end of radiation

Spinal cord compression

Treatment: - surgery is only occasionally indicated - solitary vertebral metastasis - neurological symptoms and signs progress despite radiotherapy and high dose dexamethason - vertebral body resection with anterior spinal stabilization is generally the operation of choice

Corticosteroids in palliative care

Special indications (Dexamethason 2x8mg 10-14 days):

superior vena cava syndrome

lymphadenopathy

lymphangitis carcinomatosa

obstruction of a hollow viscus (e.g. Bowel, ureter)

postradiation inflamatory

pericarditis exudative

hypercalcaemia

hormonal therapy

Corticosteroids in palliative care

Neuropathic pain

bone pain

neuropathic pain from infiltration or compression of neural structures

increased intracranial pressure

arthralgia

neuromyopathy

Corticosteroids in palliative care

Other indications:

anorexia

cachexia

difficulty with breathing

nausea, vomiting

fever