NUTRITIONAL REGULATIN OF GROWTH
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Transcript NUTRITIONAL REGULATIN OF GROWTH
NUTRITIONAL
REGULATION OF
GROWTH
ANSC 590 ANIMAL GROWTH
AND DEVELOPMENT
NUTRIENT PARTITIONING
Plane of nutrition
Homeostasis
Maintenance
Growth
Production
Repro,
milk, finishing, etc.
NUTRIENT PARTITIONING
Dietary energy
Energy density
Order of priority for energy on
physiological systems
Nervous,
circulatory, respiratory, digestive,
and reproductive
Skeletal, muscle, adipose
NUTRIENT PARTITIONING
Adipose tissues
Mesenteric
Perirenal
Subcutaneous
or intermuscular
Intramuscular or marbling
NUTRIENT PARTITIONING
Homeorhesis – longterm coordination of nutrient
partitioning
Positive energy balance
When food is plentiful to take care of maintenance, growth and
fattening
Feed efficiency is in favor of greater muscle deposition
Efficiency is in favor of lactation over dry cow
Negative energy balance – when animals don’t have
adequate energy for maintenance and production needs
Loss of weight and reduced performance
Undernutrition
Pre-natal: fetal concentration of nutrients
are greater than that of maternal plasma
Low birth weights are often due to
undernourishment during fetal
development
Table 11. 1 and 11.2
Ex.
Runt piglets
Undernutrition
Runt pigs- less weight with smaller vital
organs, less DNA in skeletal muscle with
less muscle fibers. Thus, they grow
slower, produce less muscle and deposit
fat faster or sooner and ultimately less
feed:gain ratios
Postnatal
Increased metabolic activity at the time of birth
for maintaining body temp
Glycogen and white adipose fat tissues are
called upon for energy
Colostrum is essential for energy and for
immune response
Milk production plays an integral role in plane of
nutrition versus growth
Postnatal
Weaning- ultimate stress and nutrient
change
Creep feeding
Ad libitum versus limited feed
Limited
provides less fat intake and deposition
Yet, has little effect on muscle accretion
Table 11.5
Compensatory Growth
Compensatory growth after a period of nutrient
restriction
If fed a high level of nutrition late in production
after being deprived of nutrients will deposit
more fat
If fed a low level of nutrition late in production,
then they will produce leaner carcasses
Figure 11.9 and table 11.6 & 7
Dietary Protein
Biological value
Reference
to protein quality
Even though energy is sufficient, protein is
essential for proper growth
Tables 11.8,11.9, 11.10
Yet, excess protein is metabolized for
energy and/or excreted
Dietary Protein
Ruminants utilize microbial protein to
satisfy part of protein requirements
Amino acids synthesized to ammonia,
CO2, and VFA’s (these are required for
microbial protein growth)- degradation
Nitrogen is the key for microbial protein
satisfaction (NPN can be a source)
Dietary Protein
Bypass protein – protein that escapes the
rumen
Degradable versus non-degradable prot.
Increased muscle accretion requires more
protein
Dietary Protein
Non-ruminants
Essential
versus non-essential amino acids
Limiting amino acids in basal diets
Amino acid balance/supplementation
Feed intake decreases with amino acid deficiencies
Catabolism of unused a.a.’s require energy which
reduces efficiency
Table 11.11
Dietary Energy
Requirements increase along with body size
Composition of wt. gain dictates total dietary
energy requirements and f:g ratios
Supplemental fats are added to give more
nutrient density
Non ruminants will assimilate fats to be similar
type as to the type in the diet
Soft
and oily fat due to melting point and type of fats
in the feed
Feed Additives
Ionophores- carboxylic polyether
ionophores to increase growth efficiency in
cattle
Coccidiostats
Antibiotics
Subtherapeutic
levels- changes microbes to
improve efficiency
Regulation of Protein Accretion
Alters rates of protein synthesis and degradation
Dependent upon nutritional regime versus plane
of nutrition required
Synthesis and degradation occurs at varying
rates
Synthesis is more sensitive than degradation
Amino acid and insulin supplies are important in
mediating muscle protein metabolism
Regulation of Protein Accretion
Protein accretion rates in liver appear to
be regulated by nutritional status through
changes in protein degradation rates
rather than through changes in synthesis
rates