Tissue of the teeth
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Transcript Tissue of the teeth
Pre-clinical Periodontics
Periodontitis
Dr Jamal Naim
PhD in Orthodontics
Classification of periodontal diseases
Gingivitis versus Periodontitis
Gingivitis is the inflammation of a periodontium with no
attachment loss or with previous attachment loss that
is stable and not progressing.
Periodontitis is the inflammation of a periodontium
caused by specific microorganisms resulting in
progressive destruction of the PDL and alveolar bone
(attachment loss) with pocket formation, recession or
both.
Gingivitis versus Periodontitis
Periodontitis
Chronic Periodontitis
Aggressive Periodontitis
Periodontitis as a Manifestation of Systemic
Diseases:
1.
Associated with hematological disorders
2.
Associated with genetic disorders
3.
Not otherwise specified (NOS)
Chronic Periodontitis
Also known as adult periodontitis
The most common form of periodontitis
Most prevalent in adults (about 35??????), can
occur in children
Associated with plaque and calculus
accumulation
Subgingival calculus is frequently found
Slow to moderate progression of destruction
Chronic Periodontitis
Clinical characteristics:
Microbial plaque formation
Periodontal inflammation
Loss of attachment and
alveolar bone
Chronic Periodontitis
Normal
moderate
Severe
Chronic Periodontitis
Subclassified into:
Localized chronic periodontitis (< 30% of sites involved)
Generalized chronic periodontitis (> 30% of sites
involved)
Slight chronic periodontitis 1 to 2 mm clinical
attachment loss
Moderate chronic periodontitis 3 to 4 mm of clinical
attach. loss
Severe chronic periodontitis ≥ 5 mm of clinical
attachment loss
Chronic Periodontitis/generalized
Chronic Periodontitis
Some factors cause an increase of disease
progression:
Local factors influence the plaque accumulation
systemic factors influence the host response
Environmental factors such as smoking and stress
influence also the host response
No clear evidence for genetic predisposition???
Aggressive Periodontitis
Clinically healthy patient
Rapid rate of disease progression
Absence of large accumulations of plaque and
calculus
Family history (genetic predisposition)
Diseases sites often infected with actinobacillus
actinomycetemcomitans
Abnormalities in phagocyte function
Hyperresponsive macrophages
Self arresting progression
Aggressive Periodontitis
Aggressive Periodontitis/localized
Aggressive Periodontitis/localized
Aggressive Periodontitis
Aggressive Periodontitis
Periodontitis as a Manifestation of
Systemic Diseases
Influence of host response
Confusing with other forms
Normally no major predisposing factors (plaque etc.)
are evident
Periodontitis as a Manifestation of
Systemic Diseases
Periodontitis as a Manifestation of
Systemic Diseases
Periodontitis as a Manifestation of
Systemic Diseases
Papillon lefevre syndrom
Classification of periodontal diseases
NUG: Necrotizing Ulcerative Gingivitis
Is the most common type of acute gingivitis.
It has been described since ancient Greek times,
and frequently affected troops fighting in the
trenches during WW1.
Develop quickly eg hours to days;
Usually associated with PAIN, discomfort, perhaps
swelling, fever, feeling unwell
NUG: Necrotizing Ulcerative Gingivitis
usually associated with spontaneous gingival
bleeding
require immediate attention
Other (older) names:
Trench mouth
Ulcero-membranous g.
Vincent’s gingivitis
NUG: Necrotizing Ulcerative Gingivitis
Etiology and risk factors:
Caused by specific bacterial groups:
Fusiform bacillus
spirochetes
Smoking
Poor oral health / pre-existing chronic gingivitis
Stress
HIV infection
malnutrition
NUG: Necrotizing Ulcerative Gingivitis
Signs:
Cater-like depressions at the crest of the crest of the
interdental papilla
The depressions are necrotic, covered by a gray (white
yellowish) pseudomembranous slough.
Red erythematous halo
Very severe halitosis
Spontaneously bleeding gingiva
May have fever, swollen submandibular lymph nodes
Increased salivation
NUG: Necrotizing Ulcerative Gingivitis
Generalised ANUG
Localised NUG
NUG and HIV
Non-resolving NUG after
conventional treatment
could indicate that the
patient has HIV infection
that is progressing to AIDS.
The best thing to do is
send the patient to their
general medical
practitioner to have blood
screen
NUP: Necrotizing Ulcerative periodontitis
May be an extension of NUG (different severity levels)
More commo by immuno-compromised Patients (HIV)
Clinical appearance same as NUG with
Presence of attachment loss
Interdental osseous craters
No pockets because of recessions
Etiology same as NUG
NUP: Necrotizing Ulcerative periodontitis