Pediatric Nursing
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Transcript Pediatric Nursing
Pediatric Nursing
Module 6
Caring for Children
with Alterations in
Neurosensory
Functions
Neurological Assessment
Assessment
indirect measurements
children under 2 years
normal growth and development
parameters
parents evaluation of their child
developmental milestones
history
prenatal
birth history
post natal
Neurological Assessment
Behavior
personality, affect, level of activity, social
interaction, attention span
Motor function
muscle - size, tone, strength
abnormal movements
Sensory function
discrimination of touch with eyes closed
Neurological Assessment
Cranial Nerves
Olfactory - smell
Optic - light perception
visual acuity
peripheral vision
Ocular motor - 6 cardinal
positions of gaze PERRLA
Trochlear - have child look
down and in
Trigeminal nerves - bite
down and try to
open jaw, sensation to face
Neurological Assessment
Abducens- look toward
temporal side
Facial - make a funny face or
smile
Acoustic - hearing and
balance
Glossopharyngeal - gag
reflex, taste
Vagus - uvula is midline,
swallow
Accessory - shrug shoulders
against
mild applied
pressure
Hypoglossal - move tongue
in all directions
Video - Neurological exam in
children
http://video.google.com/googleplayer.swf?docid=3314499774483652601&hl=en&fs=true
http://video.google.com/googleplayer.swf?docid=7363212422012619904&hl=en&fs=true
http://video.google.com/googleplayer.swf?docid=-728577715202828264&hl=en&fs=true
Increased Intracranial Pressure
Causes
tumors
accumulation of fluid
within the ventricular
system
bleeding
edema in cerebral
tissues
early signs and symptoms
are often subtle and
assume many patterns
Assess for signs of Increased
Intracranial Pressure
Level of consciousness (LOC)
earliest indicator of changes in
neurological status
1. Alertness
arousal-waking state
ability to respond to stimuli
2. Cognitive abilities
process stimuli
produce verbal and motor
responses
Increased Intracranial Pressure
Signs/symptoms
Lack of painful stimuli is abnormal and is
reported immediately
as ICP increases LOC decreases
3. Vital Signs
pulse
variable, may be rapid or slow, bounding or
feeble
B/P
normal or elevated with a widening pulse
pressure, at shock level
Respiration's
varies
Increased Intracranial Pressure
Signs/symptoms
Temperature
elevated especially with infections and
intracranial bleeding
subnormal in a coma of toxic origin
Pupils
size and reactivity
bilateral vs unilateral
sudden fixed and dilated pupils is a
neurosurgical emergency
pressure from herniation of the brain
through the tentorium
Neuromuscular - Signs/symptoms
Neuromuscular Movement
strength, spontaneous movements
asymmetric or absent movements
tone
may be increased or decreased
tremors, twitching, spasms
purposeless flapping
hyperactive or flaccid
Increased Intracranial Pressure
Signs/symptoms
Posturing
decorticate
adduction and
flexion
decerebrate
rigid extension and
pronation
Diagnosis Procedures
Lumbar puncture
measure pressure and sample
for analysis
Subdural tap
r/o subdural effusions,
relieves ICP
EEG
measures electoral activity
detects abnormalities
Diagnosis Procedures
Computer Tomography (CT)
visualizes horizontal and vertical
cross section of the brain
distinguishes density
MRI
permits tissue discrimination
unavailable with other techniques
Transillumination
localized glowing seen in abnormal
fluid
Diagnosis Procedures
Labs
CSF
blood glucose
electrolytes
Ca, Mg, Na
clotting studies
liver function tests
blood cultures
drug titre
Cerebral Trauma
Head Injury
Etiology
falls, MVA, bicycle injuries
head is larger, heavier
children curious
incomplete motor development
Concussion
Contusion/laceration
Fracture
Shaken Baby Syndrome
coup
countrecoup
Fatal bacterial
meningitis
Meningitis
Inflammation of the
meninges
Spread
vascular dissemination
OM or URTI
exudate covers the
brain
brain becomes
hyperemic and
edematous
Meningitis
Causative Organism
H. Influenza, type B
S. Pneumoniea
N. Meningitis
Meningococcus
Signs and Symptoms
FUO
lethargy
Meningitis
Signs/symptoms
irritable
vomiting and/or diarrhea
signs of meningeal irritation
guarding of the neck
nuchal rigidity
cries when moved
poor feeding
Meningitis
Diagnosis
Labs
CSF
culture, glucose, protein, cell count, gram
stain
Blood Culture
r/o sepsis
Urine Culture
r/o UTI
Chemistry panel
electrolytes, glucose, BUN, creatinine
Meningitis
Treatment
Antibiotics
administer within 1 hour of diagnosis
type is based on age and causative
organism
neonate - ampicillin / claforan
3 months to 3 years - ampicillin /
ceftriaxone
older children - penicillin / chloramphemicol
Meningitis
Treatment
Fluid Management
fine balance between dehydration and
cerebral edema
child may be dehydrated due to v/d, poor
po, fever
2/3 maintenance of IV replacement
fluid restriction
Meningitis
Nursing Care
PC: Neurological dysfunction
cerebral hypoxia
seizures
increased ICP
PC: Seizure
High Risk for spread of infection
needs resp. isolation for first 24 hrs of
antibiotic therapy
Meningitis
Nursing Care
Fluid Volume Deficit: less than body
requirements r/t dehydration
NPO/fluid restriction
I&O
daily weights
Labs
specific gravity and electrolytes
IV fluid - careful, conservative
replacement
Meningitis
Nursing Care
PC: Neurological damage
seizures
sequelae to meningitis
seizures
hydrocephalus
visual/hearing deficits
Reye Syndrome
Toxic encephalopathy with additional organ
involvement
Etiology
follows viral illness, ASA
Signs and Symptoms
fever
decrease LOC
hepatic dysfunction
Prognosis
good
Febrile Convulsions
Age
most common between 6 months and 3
years
Occurrence
Seizure accompanied by fever without
CNS infection
Occurs during the temperature rise
Treatment
fever - tylenol
seizure - ativan, valium
Tonic clonic seizure
Tonic – stiff
Clonic - jerking
Rescue position
Assessment
seizure precautions
emergency
treatment
rescue position
Nursing Care
protect from injury
open airway
accurately observe
and record
happenings
Hydrocephalus
Hydrocephaly
Abnormal condition characterized by an increase volume of
normal cerebrospinal fluid under increased pressure with in
the intracranial cavity
Communicating
obstruction is located in the subaranoid
cistern or within the subarachnoid space
Non-communicating
blockage is within the ventricles
Hydrocephaly - Pathology
3 possible mechanisms
leading to hydocephalus
1. Over production of
CSF
2. Defective absorption
of CSF
3. Obstruction of CSF
3 major causes
inflammation
congenital malformations
tumors
Hydrocephalus
Signs/symptoms
Signs of increased fluid pressure
tense or bulging anterior
fontanel
scalp becomes thin and shiny
vein dilate
cranial suture lines begin to
separate
Other clinical symptoms
vomiting
wide bridge between eyes
bulging eyes - sunset eyes
Hydrocephalus
Signs/symptoms
Severe Form
head size increases rapidly
infant’s cry is shrill, high pitched
hyperirritability, restlessness
Older Children
no head enlargement
papilledema
spasticity
H/A
ataxia
Alter mental status
strabismus
Hydrocephalus
Treatment
Surgical
VP (ventriculo-peritoneal)
Shunt
Nursing Care
Pre-op
assessments
daily head circumference
size and fullness of anterior
fontanel
behavior
nutrition - vomiting
Hydrocephalus - Nursing Care
fluid and electrolyte needs
positioning
prevent pressure ulcers
support the neck
good skin care
neuro assessments
LOC
irritable child/infant
vital signs
observe for seizures
Hydrocephalus
Nursing Care
Post-op
monitor feeding and behavior patterns
assess for increasing ICP and cerebral
irritability
HOB flat or set elevation
Shunt observation
infection - along the line or cerebral
abdominal girth
valve function, blockage, separation
emotional needs - hold and cuddle
teaching
Cerebral Palsy
Non-specific disorder
characterized by early onset of
movement and posture
impairments
abnormal muscle tone and
coordination
Spastic
hypertonicity, stiff
Dyskinectic
slow, worm-like movement
Spina bifia - myelomeningocele
Failure of the neural tube to
close during early development
Treatment
early surgical closure
Associated Problems
hydrocephalus
paralysis
bone deformity
Andrew, age 10 was a passenger in a MVA 3
weeks ago, he sustained a closed head injury
from the impact. He is unconscious in the E.R.
What are is needs in the Emergency Room?
What are his priority nursing interventions?
He was admitted to the PICU, now transferred
to your Pediatric Unit. He tracks his parents
movement, he is receiving 02 via trach collar,
has G-tube with enteral feedings, is incontinent
of urine and stool, is able to nod his head
appropriately.
Why do you think Andrew has a trach?
Why do you think Andrew has a G-tube?
What risk factors predispose Andrew to
infection?
Why is he on these medications?
ranitidine 70mg bid - zantac
metoclopramide 3.5 mg qid - reglan
phenytoin sodium 70mg bid - dilantin
How can you intervene to help met Andrew’s
growth and development needs?