Transcript Chapter
Medical Mycology
Prof. Khaled H. Abu-Elteen
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4223 – The Fungi
Fungal Diseases
Mycosis- fungal infection
< 100 cause human disease
Not highly contagious
Humans acquire from nature
Groups based on degree on tissue involvement
and mode of entry
Cutaneous mycoses-dermatophytes
Epidermis, hair & nails
Contagious-direct or indirect contact
Secrete keratinase that degrades keratin
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Cutaneous Mycoses
Tinea( worm) capitis –blisters with scaly ring
Ringworm of the scalp
Spreads circularly forming bald spots
Spread by contact with fomites , cats and
dogs
Tinea cruris- ringworm of groin
Tinea pedis - athlete's foot
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Systemic Mycoses
Dimorphic fungi
Yeast form is invasive
Can spread throughout body
Usually caused by fungi in soil
Inhalation of spores
Begins in lungs and spread to rest of
body
Not contagious person to person
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Introduction
Obligate Parasitic Fungi
(dermatophytes): evolved to attack the
outer surface of humans
Facultative soil fungi: thermal
dimorphic saprobes, adaptations to
human body
Opportunistic saprobes: attack people
with compromised immune systems
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Introduction
Fungal Infections
Superficial infections: involve outermost
layers of skin and its appendages [ nails or
hair] ( Dermatophytosis)
Cutaneous infections: involve deeper layers
of skin causing allergic or inflammatory
response
Subcutaneous infections: fungi with low
virulence, localized infection, or spread by
mycelial growth
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Introduction
Systemic infections: caused by true
pathogenic fungi or opportunistic
saprobes
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Mycoses: diseases cause by
fungi
Superficial
Subcutaneous
Opportunistic
Cutaneous
Systemic
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The Situation
Frequency
- most common fungal pathogen worldwide
- 4th leading causes of nosocomial infections, 40% mortality
- significant mortality and morbidity in low birth-weight infants
- affects 75% women, 45% experience recurrence
> 10 million visits/year
- classified as a STD by CDC
Immunocompromised
- cancer and HIV-AIDs patientsC
- most commonly manifested in patients with leukemia
or HIV-AIDs infections. Oral candidiasis is often a
clue to acute primary infectionC
Public Concerns
- increasing resistance to drug therapies due to antibiotics and
antifungals
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FUNGAL DISEASES
Mycosis: Any fungal disease. Tend to be chronic because fungi
grow slowly.
Mycoses are classified into the following categories:
I. Systemic mycoses: Fungal infections deep within the body.
Can affect a number if tissues and organs.
Usually caused by fungi that live in the soil and are inhaled.
Not contagious.
Examples:
Histoplasmosis (Histoplasma capsulatum): Initial
infection in lungs. Later spreads through blood
to most organs.
Coccidiomycosis (Coccidioides immites):
Resembles tuberculosis.
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Systemic Mycosis: Histoplasmosis
Disseminated Histoplasma capsulatum, lung infection.
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FUNGAL DISEASES (Continued)
II. Cutaneous mycoses: Fungal infections of the skin, hair, and
nails.
Secrete keratinase, an enzyme that degrades keratin.
Infection is transmitted by direct contact or contact with
infected hair (hair salon) or cells (nail files, shower floors).
Examples:
Ringworm (Tinea capitis and T. corporis)
Athlete’s foot (Tinea pedis)
Jock itch (Tinea cruris)
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Opportunistic Infection by Candida
albicans in an AIDS Patient
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Severe nail infection with Trichophyton rubrum in
a 37-year-old male AIDS patient.
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Disseminated Histoplasma capsulatum, skin infection.
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Cutaneous Mycosis
Ringworm skin infection: Tinea corporis
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Cutaneous Mycosis
Candida albicans
infection
of the nails.
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FUNGAL DISEASES (Continued)
III. Subcutaneous mycoses: Fungal infections beneath the
skin.
Caused by saprophytic fungi that live in soil or on vegetation.
Infection occurs by implantation of spores or mycelial
fragments into a skin wound.
Can spread to lymph vessels.
IV. Superficial mycoses: Infections of hair shafts and
superficial epidermal cells. Prevalent in tropical climates.
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FUNGAL DISEASES (Continued)
Opportunistic mycoses: Caused by organisms that are
generally harmless unless individual has weakened defenses:
AIDS and cancer patients
Individuals treated with broad spectrum
antibiotics
Very old or very young individuals (newborns).
Examples:
Aspergillosis: Inhalation of Aspergillus spores.
Yeast Infections or Candidiasis: Caused mainly
by Candida albicans. Part of normal mouth,
esophagus, and vaginal flora.
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Cutaneous Infections
Dermatophytic hyphomycetes
40 species
Epidermophyton (2 species)
Microsporum (17 species)
Trichophyton (24 species)
50% of dermatophytes human specific
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Cutaneous Infections
Cause common tinea (ringworm)
Grow only on humans
Reservoir not in soil or animals
Reservoir in carpets and upholstery
for up to two years
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Cutaneous Infections
Trichophyton
rubrum
Chronic infections
of the toe nails
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Tinea corporis
Subcutaneous mycoses
Subcutaneous infections - over 35
species produce chronic inflammatory
disease of subcutaneous tissues and
lymphatics. e.g. sporotrichosis ulcerated lesions at site of inoculation
followed by multiple nodules - caused
by a dimorphic fungus: Sporotrix
schenckii.
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Cutaneous Infections
Microsporum canis
Reservoir in cat
May move to humans or dogs
Dies out after one or two personperson transfers
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Cutaneous Infections
Disease process
Fungus stimulates epithelial cells of
skin to divide more frequently
Makes more keratin available to
fungus
Some species race specific in humans
Some species body location specific
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Cutaneous Infections
Candida albicans – candidiasis
Normal component of gut mycota
Excessive wetness overgrowth on
skin
Vaginal candidiasis common in
pregnant women
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Biology of Candida albicans
Commensal
Pathogen
A thin-walled dimorphic fungus
Morphogenesis
Unicellular yeast (harmeless)
Filamentous (pathogenic)
Principal Cell Wall Polymers
Gluccan
Mannan
Figure 1. Yeast in Oral Scraping
A sample of an oral scraping contains yeast cells and
pseudohyphae
(www.doctorfungus.org)
Strict aerobe, favors moist surfaces
Commensally found in gut, genitals, and lungs
Body Temp 37º C, neutral pH
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Rapid Multiplication & Spread
Diseases by C. albicans
Thrush
Esophagitis
Cutaneous Candidiasis
Genital Yeast Infections
Deep Candidiasis
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Oropharyngeal Thrush
* Pseudomembranous
* Atrophic
* Angular chelitis
Figure 1. Angular chelitis
Symptoms
Risk Factors
HIV
Treatment: topical
antifungals
Figure 2. Oral Thrush, atrophic
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Figure 3. Oral Thrush, pseudomembranous
Genital Yeast Candidiasis
Symptoms
Risk Factors
- disruption of normal
microbiota
Figure 1. Vaginal Yeast Culture
Treatment
- direct genital administration
- tablets, suppositories, creams
Figure 2. Plasma cell balanitis. A
band-like infiltrate of plasma cells is in
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Deep Candidiasis
Figure 1. Four forms of invasive candidiasis
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Pathogenesis
Host Recognition
Adhesins
Enzymes
Hydrolases: Phosphoplipases, Lipases, Proteinases
Morphogenesis
Yeast form to Filamentous hyphae/pseudohyphae
Phenotypic Switching
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Virulence assay of different C. albicans strains using the skin equivalent (AST 2000)
Figure 1. skin equivalent before infection
Figure 2. Infection with pathogenic clinical isolate of C. albicans.
After 48 h the yeast penetrates the skin equivalent and destroys
the tissue
Figure 3. Infection with non-pathogenic C. albicans. This strain is not
able to penetrate into the tissue and thus behaves as avirulent as shown
in the mouse model of systemic infection.
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(Fraunhofer, 2002)
MORPHOGENESIS
Figure 1. Morphogenesis.
Morphogenesis in
C. albicans is a pivotal
virulence factor that allows
rapid multiplication and
subsequent dissemination
in host tissue.
(www.kent.ac.uk)
Figure 2. Morphogenic forms of Candida albicans
http://cbr-rbc.nrc-cnrc.gc.ca/thomaslab/candida/caindex.html
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Subcutaneous Infections
Fungal Activity
Fungi normally saprobic
Introduced through wounds
Adapt to the human animal by changes in
morphology
physiology
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Subcutaneous Infections
Chromoblastosis
Common among barefoot peoples of the
tropics
Soil hyphomycete species
Enters human by thorns or wood slivers
Fungus grows host cells respond by
rapid cell division wart-like growths
on feet or legs
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Subcutaneous Infections
Mycotic Mycetoma
Disease of barefoot tropical people
Entry: wound on foot
Attacks various tissues
Stimulates formation of tumor
Compact fungal colonies form within
tumor
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Subcutaneous Infections
Skin ruptures and
some colonies
extrude
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Systemic Mycoses
Introduction
Caused by . . .
Specialized pathogens
Dimorphic
One form outside the host
Another form inside the host
Opportunistic saprobes
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Systemic Mycoses
Dimorphic Pathogen Mycoses
Histoplasmosis
Histoplasma capsulatum
Grows
on bird droppings, chicken
manure, bat guano
Conidia inhaled primary lung
infection almost always fatal until
recently
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Systemic Mycoses
Histoplasma
capsulatum
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Systemic Mycoses
Coddidioidomycosis
Coccidioides immitis
Dry, saline soils
Endemic to SW deserts of North
America
Valley fever
Until recently – almost always fatal
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Systemic Mycoses
Infection, disease process, and clinical
symptoms similar to histoplasmosis
Can be effectively treated with
fluconazole
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Opportunistic Pathogens/Disease
General
Pathogens all grow well at 37C
None cause disease in well individuals
Require breakdown in resistance system
Complication of diabetes, AIDS,
advanced cancer, sequel to steroid or
antibiotic treatments
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Opportunistic Pathogens/Disease
Zygomycosis
Species of Zygomycota
Rhizopus, Mucor, Rhizomucor
Rhinocerebral mycosis
Spores enter through sinuses
Grows rapidly outward to the eyes and
inward towards the brain
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Opportunistic Pathogens/Disease
Aspergillosis
Aspergillus sp.
Bronchiopulmonary aspergillosis
Mucus within the bronchi severe
allergic reaction
Aspergilloma
Forms a mycelia ball in lung cavity
formed from earlier TB
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Opportunistic Pathogens/Disease
Surgical intervention often required
Invasive aspergillosis
Severely debilitated
Immunosuppressed (AIDS)
Almost always fatal until recently
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AIDS and Mycoses
Aspergillosis
Candidiases (Candidiasis seen in 2/3 of
AIDS patients
Cryptococcosis
Zygomycosis
Esophogeal candidiasis and
cryptococcosis are strong indicators of
AIDS
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PRIMARY ANTI-FUNGAL
AGENTS
1.
2.
Polyene derivatives
Amphotericin B
Nystatin
Azoles
Ketoconazole
Fluconazole
Itraconazole
Voriconazole
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Azoles
There are a few rare
serious side effects
from Itraconazole and
Fluconazole
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5-fluorocytosine
(5-FC)
Interferes With RNA
Synthesis
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MECHANISMS OF ACTION
Polyenes
Azoles
Griseofulvin
5 - FC
Ergosterol in cell
membrane
Interfere with
ergosterol synthesis
Forms a barrier to
fungal growth
Inhibits RNA
synthesis
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