Transcript Slide 1

Mentoring the Mentor
Stuart White, DC, DACBN, CCN
Whole Health Associates
1406 Vermont
Houston, Texas 77006
713/522-6336
[email protected]
www.wholehealthassoc.com
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www.doctorofthefuture.org
Mentor goals:
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To declare what is possible and establish a
commitment to that possibility
Address personal and professional barriers
limiting the ability to serve
Evolution of vision/mission/ethics that drive
success
Create immediate action steps to apply
learning and growth
Construct the round table of applied
trophologists
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Mentoring the mentor:
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Who are the mentors? – Practitioners
Who are we mentoring? – Patients and
GAP
What’s the purpose? – Optimized life
How does it work? – Whatever you
learn you teach someone else (anyone
else)
Who’s is included? – Self selection, you
pick yourself
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Mentoring the mentor:
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Each participant attends monthly teleconferences
(1 hour in duration, 4th Thursday of month) creating
a round table discussion/exploration of the
dynamics and details of a nutrition-based holistic
practice
Each participant chooses how to convey the notes
and information to their world and community – no
information squandering
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Learning wisdom Never seek a lesser moment!
Review - Distinguish yourself
• It is more apparent why people are choosing alternative
health care professionals who specialize in a functional
approach
• No matter you specialty or technique you must distinguish
yourself as an expert – people are just seeking to
understand and they need you to do so
• Typically in the healthcare industry people are receiving
shallow answers that leave them puzzled with the mystery
of “Why is this happening to me?” and “ What can I do
about it?”
• Trends research over 10 years ago identified a number of
factors essential to being successful in the nutritional field –
one of those was establishing yourself as an expert
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Review - Explanation as hope
• The practitioner’s ability to explain health issues and therapeutic outcomes
creates an inflation of understanding in the patient which feels like hope
• Today in the professional world there is so much avoidance of ‘giving false
hope’ that often we end up offering little hope at all
• I propose another model that bolsters hope and expectation and subsequently
practices accountability as to whether the therapeutic endeavors are achieved or
not
• As long as the hope that has been instilled is revisited and acknowledged as
being accomplished or not the betrayal of false hope can be avoided
• So as an example, if a practitioner was describing the potential for nutritional
intervention through supplements and diet modification to improve the lipid
profile, then s/he would need to revisit to success or failure of the experiment
within a reasonable period of time
• Our community is starving for legitimate hope, as a starting place, as
empowerment to begin, as an idea to act upon
• There is genius in hope
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Seven Pillars
Unified Mechanisms
of Health
Promoting Physiology
7 Pillars of Healing
7 Unified Mechanisms of Health
Endocrine/Hormonal
Glycemic Management
pH Bioterrain
Immuno-Inflammatory
Circulatory Status
Digestive Potency
Cellular Vitality
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Endocrine
Hormonal
Cellular
Vitality
Glycemic
Management
Normal
Miracle
Digestive
Potency
Circulatory
Status
pH
Bioterrain
Minerals
Immune
Inflammatory
Practice Aging Be glad of life, because it gives you the chance to love and to
work and to play and to look up at the stars; to be satisfied
with your possessions; to despise nothing in the world except
falsehood and meanness, and to fear nothing except
cowardice; to be governed by your admirations rather than
your disgusts; to covet nothing that is your neighbor’s except
his kindness of heart and gentleness of manners; to think
seldom of your enemies, often of your friends … and to
spend as much time as you can with body and with spirit.
These are little guideposts on the footpath to peace.
Henry Van Dyke
Understanding
Detoxification
Stuart White DC, DACBN
Lee Carroll B.Sc.
Topics
1. Liver detoxification
2. Cytoprotection and Nrf2/ARE
3. Gut flora dysbiosis
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Detoxification:
“The efficient removal from the
body of chemicals likely to
effect tissues”
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Source of Toxins in the Body
1.
2.
3.
4.
5.
Environmental chemicals or contaminants
Food chemicals or contaminants
Drug therapy
Metabolism e.g. steroid hormones, bile acids
Endogenous toxins, i.e. abnormal metabolism,
dysbiosis
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Conditions Caused or Exacerbated
by Toxin Overload
 Immune deficiency
 Neurological disorders
 Chronic inflammatory disorders
 Allergies
 Autoimmune disease
 Liver damage, kidney damage etc
 Leaky gut syndrome
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Ways in which the Body Deals with
Toxins in Body Fluids
1. Storage in adipose tissue
2. Direct elimination via the kidneys, lungs etc
without further processing
3. Elimination via the urine, bile etc after
processing (biotransformation) by the liver
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Biotransformation of Toxins
 The aim of biotransformation is to render the
toxin more easily excreted
 Reactions involved in the biotransformation of
toxins by the liver can be divided into two
distinct categories:
 Phase I reactions
 Phase II reactions
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Phase I Reactions
 Largely dependent on cytochrome P450 (CYP)
enzymes
 Involves the generation of free radicals
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Phase II Reactions
 Process the by-products of phase I reactions
 Metabolites then excreted from the body
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Bioactivation
 Phase I reactions can result in the production of
more toxic substances - bioactivation
 These more toxic substances can:
 Be further metabolized by Phase II enzymes,
which renders them harmless and increases
excretion
 Cause liver damage (hepatotoxicity)
 Have a teratogenic effect
 Lead to immunological reactions
 Cause mutation by binding with DNA
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Influencing Detoxification Processes
 In order to adequately process toxins there has
to be a balance between Phase I and Phase II
enzymes
 If Phase II is overloaded or inefficient, toxic
substances from Phase I metabolism will
increase
 This increases the risk of hepatotoxicity and
tissue effects more broadly
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The Nrf2/ARE Pathway
 A new perspective on detoxification
 A basic cellular survival mechanism
 A primordial pathway, fundamental to all
animals' cells, it describes a targeted approach
to cytoprotection
 The Nrf2/ARE pathway is a dynamic response
induced by oxidative/chemical stress on the cell
that is fundamentally new and novel
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Functions of the Nrf2/ARE Pathway
 Nrf2 is expressed in all tissues, but levels vary,
with higher levels in key detoxifying organs: the
liver and kidneys
 Activation of Nrf2 activates more than 200 genes
 Nrf2 activation enhances DNA repair, heme
metabolism, efflux transport of toxins and
glutathione synthesis
 It activates detoxification, stabilizes proteins,
strengthens cellular integrity and reduces the
inflammatory response
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Functions of the Nrf2/ARE Pathway
 Key antioxidant enzymes are induced, including
catalase, superoxide dismutase (SOD),
thioredoxin, peroxiredoxin, sulfiredoxin, ferritin,
metallothionein and haem oxygenase 1 (HO-1)
 Key phase II enzymes are also induced,
including glutathione S-transferases (GSTs) and
NAD(P)H: quinone oxidoreductase 1 (NQO1)
Maher J, Yamamoto M. Toxicol Appl Pharmacol 2010; 244(1): 4-15
Lewis KN, Mele J, Hayes JD et al. Integr Comp Biol 2010; 50(5): 829-843
Itoh K, Mimura J, Yamamoto M. Antioxid Redox Signal 2010; 13(11): 1665-1678
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Nrf2/ARE in Health and Disease
Prevention
 Involvements of Nrf2/ARE in maintaining health
and preventing disease:
 Healthy aging and longevity
 Protection against radiation
 Benefits in diseases involving oxidative
damage and inflammation
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What is Gut Flora?
 A large, diverse and dynamic population of
microorganisms
 Native bacteria are acquired during birth and
during the first and second years of life
 Transient bacteria are continuously being
ingested from the environment via:
 Food
 Water
 Probiotics
Guarner F. Digestion 2006; 73(Suppl 1): 5-12
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Distribution of Gut Flora
 The stomach and duodenum have very low numbers
around 103 cfu per g of contents
 There is a progressive increase along the jejunum
and ileum from 104 → 107 cfu/g
 The large intestine contains around 1013-14 cfu/g
1014 = 100,000,000,000,000
Jones BV, 2010 A metazoan perspective: The human gut mobile metagenome, Gut
Microbes 1:6, 415-431
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Bacteria ‘R’ US
Only one out of every ten cells in our
body is human!
Bacterial
Human
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Gut Flora Diversity
 Up to 40,000 different species of bacteria
found across the human gut1
 Every person has a pattern of predominant
and subdominant species that is unique to
them2
 Some bacterial strains are unique to each
person2
1. Frank DN, Pace NR Gastrointestinal microbiology enters the metagenomics era.
Curr Opin Gastroenterol 2008; 24: 4 -10
2. Jones BV, 2010 A metazoan perspective: The human gut mobile metagenome,
Gut Microbes 1:6, 415-431 Guarner F. Digestion 2006; 73(Suppl 1): 5-12
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..
Guarner F. Digestion 2006; 73(Suppl 1): 5-12
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3 Functions of Gut Flora
Metabolic functions:
 Fermentation of nondigestible carbohydrates
which promotes the growth of the microflora and
the production of short chain fatty acids (SCFA)
 Salvage of dietary energy
 Enhanced absorption of mineral ions e.g. calcium,
magnesium and iron
Guarner F. Digestion 2006; 73(Suppl 1): 5-12
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3 Functions of Gut Flora
Metabolic functions:
 Production of some vitamins, e.g. K, B5, B7, B9,
B12
 Synthesis of amino acids from ammonia or urea
 Control of colonic pH
 Metabolism and enterohepatic circulation of
xenobiotics
Guarner F. Digestion 2006; 73(Suppl 1): 5-12
Roberfroid MB, Bornet F, Bouley C et al. Nutrition Reviews 1995; 53(5): 127-130
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3 Functions of Gut Flora
 Protective functions:
 Provide a barrier effect that prevents the
invasion of pathogens and by the excretion of
antimicrobial substances
 Trophic functions:
 Control of epithelial cell proliferation and
differentiation
 Immune system development and regulation
Guarner F. Digestion 2006; 73(Suppl 1): 5-12
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What is Dysbiosis?
 Metchnikoff (1907) was the first proponent of
probiotics. Described dysbiosis as altered
pathogenic bacteria in the gut1
 The state of disordered microbial ecology that
causes disease2
 A breakdown in the balance between protective
versus harmful intestinal bacteria 3
1
2
3
Hawrelak JA, Myers SP. Alt Med Review 2004; 9(2): 180-197
Pizzorno JE, Murray MT (eds). A Textbook of Natural Medicine, 2nd Edn, Vol 1. Churchill
Livingstone, Edinburgh, 1999, pp.110-111
Tamboli CP, Neut C, Desreumaux P et al. Gut 2004; 53: 1-4
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Common Causes of Dysbiosis
 Diet including excessive protein, excess refined
carbohydrates, lack of fiber, excess fat, sulfer
 Antibiotics
 Stress
 Decreased immune status (especially low SIgA)
 Decreased gut motility and poor digestive function
 Low hydrochloric acid production
 Intestinal infection and infestation
 Altered intestinal pH
Hawrelak JA, Myers SP. Alt Med Review 2004; 9(2): 180-197
Pizzorno JE, Murray MT (eds). A Textbook of Natural Medicine, 2nd Edn, Vol 1. Churchill
Livingstone, Edinburgh, 1999, pp 111.
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Diseases Linked to Dysbiosis
Autoimmune Diseases
 Crohn’s disease
 Ulcerative colitis
 Rheumatoid arthritis
 Ankylosing spondylitis
 Chronic active hepatitis
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Diseases Linked to Dysbiosis
Gut Disorders
 Irritable bowel syndrome (IBS)
 Flatulent dyspepsia
 Certain types of food sensitivities
 Chronic diarrhea and constipation
 Gastrointestinal infections
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Diseases Linked to Dysbiosis
Other Disorders
 Allergies such as asthma and hay fever
 Poor immunity
 Chronic skin disorders
 Insulin Resistance and Diabetes
 Neuropsychiatric problems especially autism??
 Obesity?? Depression??
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References for Previous slides
Crohn’s Disease
Rashid T, Ebringer A. Autoimmune Dis 2012; 2012: 539282
Kranich J et al. Semin Immunol 2011; 23(2): 139-145
Pöllänen R et al. J Autoimmun 2009; 32(3-4): 172-177
Mason RM et al. Br Med J 1958; 1(5073): 748-751
Blaser M. Nature 2011; 476(7361): 393-394
Plantinga TS et al. Curr Opin Pharmacol 2012 [Epub ahead of print]
Lecat A et al. Biochem Pharmacol 2010; 80(12): 2021-2031
Morgan AR et al. Hum Immunol 2012; 73(4): 416-420
Rheumatoid Arthritis
de Almeida DE et al. FEBS Lett 2011; 585(23): 3619-3626
Scott IC et al. Best Pract Res Clin Rheumatol 2011; 25(4): 447-468
Firestein GS. Nature 2003; 423(6927): 356-361
Mangat P et al. Arthritis Res Ther 2010; 12(3): 209
de Smit MJ et al. Anaerobe 2011; 17(4): 196-200
Detert J et al. Arthritis Res Ther 2010; 12(5): 218
Hoovestol RA, Mikuls TR. Curr Rheumatol Rep 2011; 13(5): 431-439
Scher JU, Abramson SB. Nat Rev Rheumatol 2011;7(10): 569-578
Ebringer A, Wilson C. J Med Microbiol 2000; 49: 305-311
Toivanen P. Ann Rheum Dis 2003; 62(9): 807-811
Scher JU et al. Arthritis Rheum 2010; 62(Suppl 10): 1390
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References for Previous slides
Ankylosing Spondylitis
Rashid T, Ebringer A. Autoimmune Dis 2012; 2012: 53928
Pöllänen R et al. J Autoimmun 2009; 32(3-4): 172-177
Mason RM et al. Br Med J 1958; 1(5073): 748-751
Ulerative Colitis
Farrell RJ, Peppercorn MA. Lancet 2002; 359(9303): 331-340
Burke DA, Axon ATR. BMJ 1988; 297: 102
Olusanya O, Steinruck H, Aleljung P et al. Zentralbl Bakteriol 1992; 276(2): 254-263
Giaffer MH, Holdsworth CD, Duerden BI. Gut 1992; 33(5): 646-650
Hartley MG, Hudson MJ, Swarbrick ET et al. Gut 1993; 34(1): 63-67
Schultsz C, Moussa M, van Ketel R et al. J Clin Pathol 1997; 50(7): 573-579
Farina C, Caprioli A, Luzzi I. Ital J Gastroenterol 1995; 27(9): 498-500
Cummings JH, Macfarlane GT, Macfarlane S. Curr Iss Intest Microbiol 2003; 4(1): 9-20
Bullock NR, Booth JC, Gibson GR. Curr Issues Intest Microbiol 2004; 5(2): 59-64
Campieri M, Gionchetti P. Gut 2001; 48(1): 132-135
Duffy M, O’Mahony L, Coffey JC et al. Dis Colon Rectum 2002; 45(3): 384-388
Irritable Bowel Syndrome
Lee RH, Pimentel M. Curr Gastroenterol Rep 2006; 8(4): 305-311
Van Citters GW, Lin HC. Curr Gastroenterol Rep 2005; 7(4): 317-320
Quigley EM. J Dig Dis 2007; 8(1): 2-7
Pimentel M et al. Ann Intern Med 2006; 145(8): 557-563
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References for Previous slides
Hepatitis
Yan AW, Schnabl B. Bacterial translocation and changes in the intestinal microbiome associated
with alcoholic liver disease, World J Hepatol. 2012 Apr 27;4(4):110-8. doi: 10.4254/wjh.v4.i4.110.
Xu M, Wang B, Fu Y, Chen Y, Yang F, Lu H, Chen Y, Xu J, Li L, Changes of fecal Bifidobacterium
species in adult patients with hepatitis B virus-induced chronic liver disease, Microb Ecol. 2012
Feb;63(2):304-13. doi: 10.1007/s00248-011-9925-5. Epub 2011 Aug 4
Ekihiro Seki and Bernd Schnabl, Role of innate immunity and the microbiota in liver fibrosis:
crosstalk between the liver and gut J Physiol 590.3 (2012) pp 447–458
Allergy, Atopic Dermatitis
O'Regan GM et al. J Allergy Clin Immunol 2009; 124(3 Suppl 2): R2-R6
Caubet JC, Eigenmann PA. Immunol Allergy Clin North Am 2010; 30(3): 289-307
Schäfer T. Curr Opin Allergy Clin Immunol 2008; 8(5): 418-422
Ionescu JG. J Med Life 2009; 2(2): 146-154
Dokmeci E, Herrick CA. Semin Cutan Med Surg 2008; 27(2): 138-143
Feary J et al. Allergy 2011; 66(4): 569-578
Rook GA, Brunet LR. Gut 2005; 54(3): 317-320
Vael C, Desager K. Curr Opin Pediatr 2009; 21(6):794-800
Gut Disorders
Kaur N, Chen C-C, Luther J and Kao JY, Intestinal dysbiosis in inflammatory bowel disease Gut
Microbes 2:4, 211-216; July/August 2011
Guarner F. Digestion 2006; 73(Suppl 1): 5-12
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References for Previous slides
Immunity
Hsin-Jung Wu, Eric Wu, The role of gut microbiota in immune homeostasis and autoimmunity Gut
Microbes 3:1, 4–14; January/February 2012
Molloy MJ, Bouladoux N, Belkaid Y, Semin Immunol. Intestinal microbiota: shaping local and
systemic immune responses, 2012 Feb;24(1):58-66
Metabolic
Patrice D. Cani* and Nathalie M. Delzenne, The Role of the Gut Microbiota in Energy Metabolism
and Metabolic Disease Current Pharmaceutical Design, 2009, 15, 1546-1558
E.O. Petrof, E.C. Claud, G.B. Gloor and E. Allen-Vercoe, Microbial ecosystems therapeutics: a new
paradigm in medicine? Beneficial Microbes, March 2013; 4(1): 53-65
Neuropsychiatric
Bienenstock J, Collins S. 99th Dahlem conference on infection, inflammation and
chronic inflammatory disorders: psycho-neuroimmunology and the intestinal
microbiota: clinical observations and basic mechanisms. Clin Exp Immunol. 2010 Apr;160(1):8591
E.O. Petrof, E.C. Claud, G.B. Gloor and E. Allen-Vercoe, Microbial ecosystems therapeutics: a new
paradigm in medicine? Beneficial Microbes, March 2013; 4(1): 53-65
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Spring Cleaning
Cleanse | Rebalance | Revitalize
Lee Carroll B.Sc.
Spring Cleaning
 Support normal liver detoxification function
 Support the body’s normal cytoprotective
mechanisms
 Promote healthy and balanced gastrointestinal
flora
1
Spring Cleaning Products




LivCo®
ChelaCo
Cruciferous Complete™
Gut Flora Balance Program
 Gut Flora Complex
 Prebiotic Inulin
 ProSynbiotic
 Vitanox®
2
Liver Detoxification
Processes
 Herbs that support Phase I without having a
similar effect on Phase II are generally not
desirable
 Should aim at supporting both Phase I and
Phase II simultaneously
 There is still much that we do not understand
with regard to liver detoxification mechanisms
and substances that influence them
 An approach involving the buffer of “chemical
complexity” is desirable
3
LivCo
Schisandra fruit 6:1 ext
from Schisandra chinensis fruit 1.0 g
167 mg
Rosemary leaf 5:1 ext
from Rosmarinus officinalis leaf 500 mg
100 mg
Milk Thistle seed 70:1 ext
30 mg
from Silybum marianum seed 2.1 g
containing flavanolignans calc. as silybin 24 mg
Suggested use: 1 tablet 3 - 4 times daily
4
LivCo
 Aid in the elimination of toxins and cleanse
the liver
 Support healthy liver function
and tissue integrity
 Protect liver tissue by supporting
normal cellular defenses
 Provide antioxidant activity
 Support and maintain cellular health
 Stimulate the biosynthesis of
protein and liver glycogen
 Support digestive health
 Ease the effects of everyday tension and stress
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Rosemary
 Rosemary contains the antioxidant activity
molecules carnosol and carnosic acid
 Both of these are now understood to be potent
primers of the Nrf2/ARE pathway
 For example both compounds have demonstrated
potential neuro-supportive activity mediated by
this pathway, which reflects on its traditional use
for memory1,2
 Recently a single 750 mg dose of Rosemary herb
improved speed of memory and alertness in
healthy older adults3
1
2
3
Kosaka K et al. J Biochem 2010; 147(1): 73-81
Martin D et al. Biol Chem 2004; 279(10): 8919-8929
Pengelly A et al. J Med Food 2012; 15(1): 10-17
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Milk Thistle
 Milk Thistle is the most important of all the liver
herbs
 Supports normal liver function and normal liver
repair processes
 Stabilizes hepatic cell membranes
 Supports normal hepatic and portal blood flow
 Its powerful antioxidant action helps protect the
liver from the damaging effects of free radicals,
generated by toxins and our own body
processes
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ChelaCo
Hawthorn herb flowering tops 3:1 ext
from Crataegus monogyna
herb flowering tops 300 mg
Containing vitexin-2-rhamnoside 2.0 mg
100 mg
Milk Thistle fruit 70:1 ext
100 mg
from Silybum marianum fruit 7.0 g
Containing flavanolignans calc. as silybin 80 mg
Garlic (Allium sativum) bulb powder
Containing alliin 2.0 mg
100 mg
Suggested Use: 1 tablet 2 - 3 times daily with meals
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ChelaCo
 Encourage the healthy function of the organs of
elimination (particularly the normal flushing of
toxins from the tissues)
 Support the body's natural defenses
against environmental insult
 Help maintain healthy blood
and tissues
 Protect liver tissue by supporting
normal cellular defenses
 Provide antioxidant activity
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Cruciferous Vegetables
 Research demonstrates that cruciferous
vegetables, such as kale and Brussels
sprouts, contain important phytonutrients
that help protect against free radicals, the
highly unstable molecules that can damage
cells and genetic material
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Cruciferous Complete
Each capsule contains:
Kale
300 mg
Brussels sprouts
300 mg
Vitamin K
4 mcg
Potassium
10 mg
Suggested Use: 2 capsules per day
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Cruciferous Complete
 Supports healthy liver function
 A source of vitamin K, which supports calcium
absorption, blood clotting, and healthy liver
function
 Provides antioxidant activity
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GI Flora Balance Program




Gut Flora Complex
Prebiotic Inulin
ProSynbiotic
Vitanox
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Gut Flora Complex
Anise (Pimpinella anisum) fruit essential oil
125 mg
Andrographis herb 10:1 extract
from Andrographis paniculata herb 1.0 g
Containing andrographolide 10 mg
100 mg
Phellodendron stem bark 20:1 extract
80 mg
from Phellodendron amurense stem bark 1.6 g
Containing berberine 36 mg
Oregano (Origanum vulgare) leaf essential oil 75 mg
Suggested Use: 3 - 6 enteric capsules per day
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The Rationale
 The GI Flora Balance program is a holistic
approach which cleanses at the same time
as promoting balance between native
bacterial colonies
 Microbial ecology moves toward a healthy
balance
 Prebiotics are essential to this process
 Consider Prebiotic Inulin as a “Colonic Nutrient”
 GI flora need specific colonic nutrients (soluble
fiber) which may be deficient in the diet
 Appropriate and individualized dietary
advice is important for successful outcomes
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The Rationale
 Probiotics have a valuable role to play
 Probiotics used alone are beneficial however we
need to recognize the limitations
 Probiotics are transient. Probiotics do not
cleanse
 Probiotic foods contain lactic acid. Lactic acid is
cleansing
 ProSynbiotic is a unique combination of probiotic
organisms and colonic nutrients (soluble fiber)
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GI Flora Balance Protocol
Every day for 6 weeks
 Gut Flora Complex,
1 capsule twice per day
 Prebiotic Inulin,
1 teaspoon twice per day
 Both taken at the same time
As required include:
 Vitanox, 2 to 3 tablets per day
 ProSynbiotic, 3 capsules per day
(At a different time to the Gut Flora Complex.
Separate by at least 2 hours.)
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GI Flora Balance
Program Benefits








Promotes healthy and balanced intestinal flora
Cleanse the lower gastrointestinal tract
Maintains a healthy GI environment
Useful as an adjunctive treatment for
gastrointestinal challenges
Support healthy digestion, improve nutrient
absorption
Support healthy immune system function
Maintains healthy skin
Promote vitality and stamina
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Spring Cleaning Program
Every day for 6 weeks
 LivCo,2 tablets twice daily
 Cruciferous Complete, 1 capsule twice daily
 Gut Flora Balance Program
 ChelaCo, 1 tablet 2 - 3 times daily with meals if
required
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Acknowledgements
Special thanks to
Professor Kerry Bone
Principles at work
• Detoxification
is vital to age-related decline and
longevity objectives
• The high density effect of research creates more
and more commitment to this lifestyle as a sound
way to live life and reduce morbidity
• Every patient must be engaged in sequential and
recurrent detoxification events to reduce the toxic
burden associated with health status
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Primary Products
• lSequential detox:
•Livco
•Livton
•Betacol
•Vitanox
•A F Betafood, Choline, Collinsonia
• Nrf2 support to incerase sirtuin and glutathione
•Herbavital
•Vitanox
•Cruciferous
• Gut Flora Reboot:
•Gut Flora Complex
•Prosynbiotic
•Zymex II, Multizyme, Wormwood
•Zymex
•Cataplex AC
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Sequential Intervention
 By giving hope through discussion of therapeutic rationale and then
accountably determine if the therapy had efficacy it is possible to initiate
activity that may assist a person to make the changes that result in healing
 Sequential intervention and accountable follow-up can show what has
worked and what may still need to be employed
 Promote an understanding of glycemic management and show the effect of
corrective management
 Allow every condition to become a strategic consideration of possible
etiology and therapeutic rationale – people are in search of experts – reveal
yourself
 The comprehensive nature of nutritional therapy means there is always more
physiology to optimize and support leaving an individual constantly refining
as long as they wish to further improve their status
 If the practitioner is accountable s/he will be allowed to experiment with
reasonable ideas
Change the world
It wants to
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