Transcript Chapter 12
Chapter 12
B-Cell Activation and Differentiation
Dr. Capers
Kindt • Goldsby • Osborne
Kuby IMMUNOLOGY
Sixth Edition
Chapter 11
B-Cell Generation, Activation,
and Differentiation
Copyright © 2007 by W. H. Freeman and Company
B cell activation
B cell encounters specific antigen
B cell presents to T helper cell
Cytokines are released for full B cell
activation
Proliferation, some of the B cells
become plasma cells
Some of the B cell clones move to
germinal centers of lymph nodes,
somatic hypermutation can occur
Class switching occurs
B cell Activation
Thymus-dependent (TD) antigens
B cell required direct contact with TH cell
B-2 B cells, majority of B cells
Thymus-independent antigens (TI)
These antigens activate B cells by pattern recognition
receptors (bacteria that might be in high amount)
Type I (TI-1) – lipopolysaccharide
Type 2 (TI-2) – highly repititous molecules (bacterial
flagella)
B cell Activation
Membrane bound
antibody have short
cytoplasmic tails
○ Too short to generate
signal by associating
with tyrosine kinases
and G proteins
Membrane Ig must
be associated with
B-cell receptor
Ig-α/Ig-β
ITIM
(immunoreceptor
tyrosine inhibitory
motif)
○ Associated with
CD22
○ Functions to
deactivate B cells –
negative regulation
○ Important in
preventing
autoimmunity
TH cells play
essential role in B
cell repsonses
TEM of interaction between B cell and T
cell
Humoral Response – Primary vs Secondary
Hapten-carrier conjugates
Hapten – low molecular weight molecule
that won’t itself induce a humoral
response
Must be coupled to suitable carrier
In vivo sites for induction of humoral responses
Blood-bourne antigen is filtered by
spleen
Antigen from tissue spaces filtered by
lymph nodes
○ Antigen either enters alone or with antigen-
transporting cells
- Langerhans cells
- Dendritic cells
○ Encounters antigen-presenting cells
- Dendritic cells
- Macrophages
- Follicular dendritic in follicles and germinal centers
T cells are green and B cells are red
Germinal centers arise within 7-10 days
after initial exposure to thymusdependent antigen in lymph node
○ 3 events in germinal centers
Affinity maturation
- Result of somatic hypermutation
Class switching
Formation of plasma and memory B cells
Cellular events in germinal centers
Dendritic cell presents antigen
to developing B cells to see
which B cells are producing
antibody with high-affinity
for that antigen
Class Switching
Dependent on cytokines to
switch from IgM to other
isotype
Thymus-dependent
antigens
Interaction of CD40 on B
cell and CD40L on T cell
X-linked hyper-M
syndrome
○ TH cells don’t express
CD40L, patients only
produce IgM
No memory cell
populations, no germinal
centers
Regulation
Humoral and cell-mediated branches must be heavily
regulated
Cytokines play important role
Antigenic competition
Previous encounter with antigen can render animal
tolerant or may result in formation of memory cells
Presence of antibody can suppress response to antigen
Some vaccines are given to babies after maternal IgG
(that was transferred across placenta) has left system
Vaccination before this will prevent proper response
and development of long-lasting memory cells