ATLS - Head Trauma modified

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Transcript ATLS - Head Trauma modified

Committee on Trauma Presents

Head Trauma

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Objectives

Describe basic intracranial physiology.

Recognize the importance of limiting secondary brain injury.

Perform a focused neurologic exam.

Stabilize and arrange for definitive care.

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Anatomy and physiology effects?

Rigid, nonexpansile skull filled with brain, CSF, and blood

CBF autoregulation

Autoregulatory compensation disrupted by brain injury

Mass effect of intracranial hemorrhage

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Monro-Kellie Doctrine Venous Volume Art.

Vol.

Brain CSF 75 mL Ven.

Vol.

Art.

Vol.

Brain Mass CSF Arterial Volume Brain Mass CSF 75 mL

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Volume – Pressure Curve

60 55 50 45 40 35 30 25 20 15 10 5-

ICP

(mm Hg)

Point of Decompensation Compensation Volume of Mass Herniation

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Intracranial Pressure (ICP)

  

10 mm Hg > 20 mm Hg > 40 mm Hg = = = Normal Abnormal Severe

Many pathologic processes affect outcome

Sustained

ICP leads to

outcome brain function and

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Cerebral Perfusion Pressure* Normal Cushing’s Response Hypotension MBP – ICP = CPP 90 10 80 100 50 20 20 80 30 * CPP

Cerebral Blood Flow

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Autoregulation

If autoregulation is intact, CBF is maintained with a mean BP of 50 to 160 mm Hg.

Moderate or severe brain injury: Autoregulation often impaired

Brain more vulnerable to episodes of hypotension

secondary brain injury

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Mild Brain Injury

GCS Score = 14–15

History

Exclude systemic injuries

Neurologic exam

X-rays as indicated

Alcohol / drug screens as indicated

Liberal use of head CT Observe or discharge based on findings

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Moderate Brain Injury

GCS Score = 9–13

Initial evaluation same as for mild injury

Admit and observe

Frequent neurologic exams

Repeat CT scan

CT scan for all

Deterioration: Manage as severe head injury

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Severe Brain Injury

GCS Score = 3–8

Evaluate and resuscitate

Intubate for airway protection

Focused neurologic exam

Frequent reevaluation

Identify associated injuries

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Classifications of Brain Injury By Morphology: Brain Focal

Epidural (extradural)

Subdural

Intracerebral Diffuse

Concussion

Multiple contusions

Hypoxic / ischemic injury

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Diffuse Brain Injury

Mild concussion

Severe, ischemic insult Normal CT Diffuse Injury

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Contusion / Hematoma

Coup / contracoup injuries

Most common: Frontal / temporal lobes

CT changes usually progressive

Most conscious patients: No operation

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Contusion / Hematoma Large frontal contusion with shift

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Epidural Hematoma

Associated with skull fracture

Classic: Middle meningeal artery tear

Lenticular / biconvex

Lucid interval

Can be rapidly fatal

Early evacuation essential

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Epidural Hematoma Temporal Epidural Hematoma Uncal herniation

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Subdural Hematoma

Venous tear / brain laceration

Covers cerebral surface

Morbidity / mortality due to underlying brain injury

Rapid surgical evacuation recommended, especially if > 5 mm shift of midline

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Subdural Hematoma

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Priorities

ABCDE

Minimize secondary brain injury

 

Administer O 2 Maintain blood pressure (systolic > 90 mm Hg)

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Focused Neurologic Exam?

GCS Score

Pupils

Lateralizing signs Consult neurosurgeon early

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Indications for CT Scan?

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Medical Management

Intravenous fluids

Euvolemia

Isotonic

Controlled ventilation

Goal: Paco 2 at 35 mm Hg

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Medical Management

Mannitol

Use with signs of tentorial herniation

Dose: 1.0 g / kg IV bolus

Consult with neurosurgeon first

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Medical Management

Other medications

Anticonvulsants

Sedation

Paralytics

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Surgical Management Scalp Injuries

Possible site of major blood loss

Direct pressure to control bleeding

Occasional temporary closure

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Surgical Management Intracranial Mass Lesion

May be life-threatening if expanding rapidly

Immediate neurosurgical consult

Hyperventilation / Mannitol

Damage control craniotomy: Transfer to neurosurgeon (rural / austere areas)

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Summary: What should I do?

Maintain mean BP > 90 mm Hg

Maintain Paco 2 near / at 35 mm Hg

Use isotonic solution for euvolemia

Frequent neurologic exams

Liberal use of CT scans

Early neurosurgical consult

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Summary: What should I not do?

Allow patient to become hypotensive

Over-aggressively hyperventilate

Use hypotonic IV fluids

Use long-acting paralytics

Paralyze before performing complete exam

Depend on clinical exam alone

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